約 3,988,327 件
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キャラ紹介 キャラ名しおばたー 現在の各クラスの状況 ハンター ファイター ブレイバー レンジャー ガンナー フォース テクター 55Lv 60Lv 60Lv 30Lv 12Lv 23Lv 1Lv 一言 8/28に入りましたsora432です、メインキャラはしおばたーになります サブキャラでななえすがいますが基本的にこのキャラしか動かしません。 チームオーダーなどもコツコツと消化していきますのでよろしくおねがいします。 ブレイバーに続きファイターがカンストし、今はハンターのカンストを目指しています。 ;
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SOR... Sorensen, Janet. 2000. The Grammar of Empire in Eighteenth-Century British Writing. Cambridge Cambridge University Press. Sorlin, Sandrine. 2018. "The Rolling Stones Promoting Monty Python The Power of Irony and Banter", in The Pragmatics of Irony and Banter, ed. Manuel Jobert Sandrine Sorlin, pp. 195-210. Amsterdam John Benjamins.
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Decoration 名前 Population Range Cost 必要条件 サイズ WelomeSign 10 2 S1000 最初から 1x1 Billboard 15 2 S1200 最初から 2x1 WhiteFlowerbed 6 2 S600 最初から 1x1 RelayTower 6 2 M150 最初から 1x1 GreenTree 6 2 S650 最初から 1x1 BeachVolleyballCout 9 2 S2500 最初から 3X2 Treehouse 30 2 D14 最初から 1x1 HotDogStand 10 2 S1200 最初から 1x1 OrangeTree 8 2 S800 最初から 1x1 CobblestonePatch 2 5 S500 最初から 1x1 ATM 18 2 S1600 最初から 2x1 GiantTrampoline 18 3 S5000 最初から 2x2 PinkTree 33 3 D16 最初から 1x1 "Capitalism" 150 5 D299 最初から 2x1 PinkFlowerbed 33 3 D16 最初から 1x1 SuperHighDive 36 4 D16 最初から 2x2 BlueFlowerbed 10 2 S1200 最初から 1x1 WeightliftingArena 25 3 S9000 最初から 1x1 TacoTruck 18 2 M600 最初から 1x2 Playground 24 2 S2500 最初から 2x2 RockClimbingTerrace 36 4 D22 最初から ? FoodTruckCorner 33 3 S18 最初から 1x2 CherryBlossomTree 30 3 D17 最初から 1x1 martialArtsTatami 30 4 S1500 最初から 3x2 OrangeFlowerbed 14 2 人口400 1x1 PineTree 15 2 人口500 1x1 FerrisWheel 38 3 人口650 ? ConstructionCrane 20 2 人口700 1x1 Clotheslines 18 2 人口800 2x1 PizzaTruck 25 2 人口1500 1x2 Gazebo 33 2 人口900 1x1 Grove 24 2 人口1000 2x2 LampholderStatue 25 2 人口1100 2x1 yellowFlowerbed 16 2 人口1200 1x1 OldShippingCrane 45 3 人口1300 ? GiantRedwood 23 2 人口1450 1x1 GraffitiWall 26 2 人口1600 2x1 Plaza 24 2 人口1800 1x1 BellTower 36 2 人口2000 1 AtlasStatue 25 2 人口2100 1 Graveyard 18 3 人口2200 4 BigGraveyard 20 3 人口2500 ? PalmTree 26 2 人口2750 1x1 Promenade 20 3 人口3000 1x1 GiantScreen 30 2 人口3500 1x2 CampingArea 22 3 人口4000 3x2 Statue 15 3 人口4200 1x1 WaterTower 27 2 人口4800 1x1 BasketballCourt 45 2 人口5400 ? PublicPool 32 2 人口6000 2x2 CylinderGarage 53 3 人口7000 ? Orchard 32 2 人口8000 2x2 Amphitheater 35 2 人口9000 2x2 TheArch 38 3 人口10000 ? TennisCourt 32 2 人口11000 1x2 "MeritsofIndustry" 36 2 人口11500 ? SoccerPitch 36 3 人口12000 ? StreetFair 35 2 人口13000 ? ExoticFruitorchard 36 2 人口14000 ? DoggyPark 36 2 人口15000 ? HedgeMaze 25 3 人口16000 ? BikeRack 18 3 人口17000 ? HotAirBalloonRide 30 4 人口18000 ? DrivingRange 30 4 人口19000 ? WarMonument 22 3 人口20000 ? PEgyptianStatue 68 3 人口21000 ? DinoSkeleton 45 4 人口22000 ? CityLake 32 3 人口23000 ? SatelliteDish 22 3 人口24000 ? FroyoHut 28 3 人口25000 ? ArcheryRange 53 4 人口26000 ? HeartStatue 28 3 人口27000 ? PerpetualWaterfallMill 54 4 人口28000 ? TickerBillboard 30 3 人口29000 ? LargeFountain 32 3 人口30000 ? BoogiePlaza 44 3 人口31000 ? CherryOrchard 44 3 人口33000 ? LemonadeStand 20 3 人口34000 ? MakeoutPoint 48 4 人口35000 ? ChampionshipTorch 27 3 D15 最初から 1x1
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TOXICOLOGICAL PROFILE FOR WHITE PHOSPHORUS -index 2 HEALTH EFFECTS 2.2 DISCUSSION OF HEALTH EFFECTS BY ROUTE OF EXPOSURE 2.2.2 Oral Exposure -3 2.2.2 Oral Exposure(2.2.2.0 preface) 2.2.2.1 Death 2.2.2 Oral Exposure -22.2.2.2 Systemic Effects 2.2.2 Oral Exposure -32.2.2.3 Immunological and Lymphoreticular Effects 2.2.2.4 Neurological Effects 2.2.2.5 Reproductive Effects 2.2.2.6 Developmental Effects 2.2.2.7 Genotoxic Effects 2.2.2.8 Cancer 2.2.2.3 Immunological and Lymphoreticular Effects There is limited information on the immunotoxicity of white phosphorus; however, there is some information that suggests that the immune system may be a target. Thymic hemorrhages were observed in two young children accidentally ingesting white phosphorus-containing fireworks (Dwyer and Helwig 1925; Humphreys and Halpert 1931). In one of these children, hyperplasia of lymphoid tissue in the intestinal wall and abdominal lymph nodes and hyperplastic lymphoid corpuscles in the spleen were observed (Humphreys and Halpert 193 1). Decreases in leukocyte levels were reported in a number of case reports involving acute ingestion of rat poison or fireworks containing white phosphorus (Diaz-Rivera et al. 1950; Ehrentheil 1957; Fletcher and Galambos 1963; McCarron et al. 1981; Newburger et al. 1948; Pietras et al. 1968). A decrease (Pietras et al. 1968) or an increase in the percentage of polymorphonuclear leukocytes (neutrophils) (McCarron et al. 1981) were also observed in individuals ingesting white phosphorus. Because the individuals vomited shortly after ingesting the white phosphorus and/or received gastric lavage, doses could not be estimated. In workers exposed to an unknown level of white phosphorus via inhalation, oral, and dermal routes, a decrease in leukocyte levels was observed (Ward 1928). No studies were located regarding immunological or lymphoreticular effects in animals after oral exposure to white phosphorus. 2.2.2.4 Neurological Effects A number of case reports of individuals accidentally or intentionally ingesting a single dose of white phosphorus have reported neurological effects. Nonspecific neurological effects including lethargy (Dathe and Nathan 1946; Fletcher and Galambos 1963; McCarron et al. 1981; Rao and Brown 1974; Rubitsky and Myerson 1949; Simon and Pickering 1976; Talley et al. 1972), sleepiness (Dwyer and Helwig 1925; Ehrentheil 1957; McCarron et al. 1981; McIntosh 1927), irritability (McCarron et al. 1981), restlessness (Diaz-Rivera et al. 1950; Ehrentheil 1957; Harm and Veale 1910), and hypoactivity (Humphreys and Halpert 1931) have been observed. Other symptoms of neurotoxicity that have been observed include coma or semi-coma (Caley and Kellock 1955; Ehrentheil 1957; Hann and Veale 1910; McCarron et al. 1981; McIntosh 1927; Wechsler and Wechsler 1951), toxic delirium and psychosis (Diaz-Rivera et al. 1950), hemiplegia (Humphreys and Halpert 1931; McCarron et al. 1981), abnormal reflexes (Wechsler and Wechsler 1951), hyperesthesia (Humphreys and Halpert 1931), coarse muscle fasciculations (Caley and Kellock 1955), unresponsiveness to painful stimuli (Simon and Pickering 1976), and marked asterixis (flapping tremor) (Greenberger et al. 1964). In addition to these overt signs of neurotoxicity, histological damage in the brain was observed in four individuals ingesting a single dose of white phosphorus. Based on this limited information, the types of cellular damage can be grouped into four categories (1) cellular changes resulting from ischemic damage found in the Purkinje cells and cerebral cortical cells of the second and third layer of the cortex (Wertham 1932); (2) direct white phosphorus-induced cellular damage to the dentate nucleus and inferior olives (Wertham 1932); (3) fatty infiltration in the ganglion cells of the cortex, neuroglial cells, Golgi cells of the cerebellum, and the cells in the pia-arachnoid space (Humphreys and Halpert 1931; Wertham 1932); and (4) cerebral edema (Rao and Brown 1974). It is not known if the cerebral edema observed in this one individual was secondary to the other types of damage. A child treated with 0.083 mg/kg/day white phosphorus for an intermediate duration became lethargic 3 months after beginning treatment and remained lethargic until treatment was discontinued .70 days later. Following cessation of treatment, the child recovered very rapidly (Sontag 1938). Overt signs of neurotoxicity were observed in a cat ingesting a single lethal dose (Fry and Cucuel 1969) and in pregnant rats exposed to a lethal dose (0.075 mg/kg/day) of white phosphorus for an intermediate duration (effects only observed during late gestation of parturition) (Bio/dynamics 1991). Tonoclonic convulsions, increased salivation and weakness were observed in the cat (Frye and Cucuel1969), and tremors were observed in pregnant rats (Bio/dynamics 1991). In another developmental toxicity study (IRDC 1985), no signs of neurotoxicity were observed in pregnant rats. All LOAEL values from each reliable study for neurological effects in each species and duration category are recorded in Table 2-2 and plotted in Figure 2-2. Because vomiting occurred or the individuals received gastric lavage shortly after ingestion, reliable dose estimations could only be made for one individual acutely exposed to 2 mg/kg/day white phosphorus (Hann and Veale 1910). 2.2.2.5 Reproductive Effects Extensive uterine hemorrhaging was observed in a 2-month pregnant woman following the intentional ingestion of 2 mg/kg white phosphorus in rat poison (Hann and Veale 1910). Autopsy results showed that the uterus was enlarged containing a hemorrhagic mole, which was consistent with a 2-month pregnancy. No effects on reproductive performance or histological alterations in the ovaries, uterus, testis, or epididymis were observed in rats administered 0.075 mg/kg/day or less in a one-generation reproduction study (Bio/dynamics 1991; IRDC 1985). The highest NOAEL value and all LOAEL values from each reliable study for effects in each species and duration category are recorded in Table 2-2 and plotted in Figure 2-2. 2.2.2.6 Developmental Effects A healthy infant was administered phosphorized cod liver oil (reported to contain 1.1 mg “pure” phosphorus per fluid ounce) from ages l-7 months (Sontag 1938). The phosphorized cod liver oil was apparently administered for the prevention of rickets. The time-weighted average dose for the 6-month exposure was 0.083 mg/kg/day. During the first 3 months of treatment, the child appeared clinically normal and grew at a normal rate. From the ages of .4 to 6 months, the child became clinically ill, gained essentially no weight, and the rate of growth in height decreased from .0.1 to 0.04 cm/day. Following replacement of the treatment with normal, nonphosphorized cod liver oil, the child appeared to recover quickly, and began to grow at a normal rate. Radiograms taken at 6 months of age showed bands of increased density at the end of all the long bones with increased thickness and density also observed in the zones of calcification. Radiograms taken between 9 months and 5 years of age showed bands of increased density in the diaphyses of the long bones, and in the pelvic, metacarpal, and metatarsal bones. This study describes formation of “phosphorus” bands of increased density in the ends of long bones and possible decreased growth in a child exposed to 0.083 mg/kg/day phosphorus for 6 months (Sontag 1938). It should be noted that radiologic densities are common at the growing points of long bones in children. However, lead poisoning, administration of nickel, certain chronic diseases like anemia, and hypervitaminosis D may also produce bands in the ends of bones, but these are much thicker and heavier (Sontag 1938). A child with Perthes’ disease was administered 0.056 mgkg/day of phosphorus for two periods of intermediate duration, separated by a period with no exposure (Phemister 1918). “Phosphorus” bands of increased density developed in the ends in the tibia, fibula, and femur during the two exposure periods, without any improvement in the child’s condition. A male child with dyschondroplasia was administered 0.026 and 0.046 mg/kg/day white phosphorus for 3 and 8 months, respectively. “Phosphorus” bands of increased density developed in the tibia, fibula, and femur. The density and thickness of the bands were greater at the high-dose level and longer-treatment period. A male child with osteogenesis imperfecta was administered 0.078, 0.063, and 0.059 mg/kg/day phosphorus for 26,3, and 18 months, respectively, separated by a period of time with no white phosphorus exposure. Treatment with white phosphorus produced marked changes, including bands of increased density at the ends of bones and increased transverse diameters of the shafts of bones in the legs and arms (Phemister 1918). Four children with moderate to severe cases of rickets were treated orally with 0.110-0.158 mg/kg/day white phosphorus for durations ranging from 64 to 149 days (Compere 1930a). “Phosphorus” bands of increased thickness and density were observed in the long bones of 1 of 2 of the children examined. An arachitic child was treated with 0.119 mg/kg/day white phosphorus for 82 days (Compere 1930b). Following treatment, the child had a “heavy phosphorus line” and increased density of cortices. Treatment with white phosphorus did not generally improve the condition of the bones in children with rickets. Because these children were sickly, the relevance of the observed effects to potential effects of white phosphorus in normal, healthy children could not be ascertained. Young, growing rabbits exposed to 0.3 mg/kg/day white phosphorus given as a pill for an acute duration had transverse bands of increased density in metaphyseal regions of the tibia and fibula, compared to a control group (Adams 1938a). However, the percentage of calcium and phosphorus, and the calcium/phosphorus ratio in the metaphyseal and cortical regions of the right tibia was similar between treated and control animals. Young, growing rabbits exposed to 0.3 mg/kg/day white phosphorus given as a pill for an intermediate duration had average growth of the tibia of 0.27 mm/day, compared to 0.36 mm/day in the control group; however, no statistical analysis of the results was reported (Adams and Samat 1940). One rabbit had histological abnormalities in the tibia including decreased size of epiphyseal cartilage plate, as well as increased density in the metaphyseal zone with trabeculae that were greater in number and extended further into the diaphysis to a greater extent, compared to a control rabbit. The trabeculae were associated with a greater amount of calcified cartilage matrix. These effects probably resulted from a decrease in the normal rate of bone resorption during bone growth, resulting in decreased rate of growth of the tibia. Weanling rats exposed to 1.25 mg/kg/day white phosphorus in the feed for an intermediate duration had widening of the metaphyseal trabeculae, broadened metaphysis, and a slightly convex lateral contour of the proximal tibia, compared to a control group (Whalen et al. 1973). Osteocytes were small and elongated compared to those in the control group, and osteocytic osteolysis and chondrolysis were decreased or missing. In the treated rats, metaphyseal trabeculae extended deeper into the diaphysis than in the controls. These effects probably resulted from decreased bone resorption during bone growth, resulting in widening trabeculae and a denser metaphysis. Very similar results were observed in studies on growing rats (Adams and Sarnat 1940) and rabbits, but not in an adult rabbit (Adams 1938b). In rats, the doses varied from 0.002% to 0.05% yellow phosphorus (Adams and Sarnat 1940) and in rabbits, from 0.6 to 6 mg (Adams 1938b; Adams and Samat 1940). A decrease in the number of viable pups and an increase in the number of stillborn pups was observed in the F1a and F1b offspring of rats exposed to 0.075 mg/kg/day; however, the incidence was not significantly (p 0.05) different from controls (IRDC 1985). These effects were not seen in a similarly designed reproduction study in which rats were administered 0.075 mg/kg/day (Bio/dynamics 1991). Neither of these studies found any significant differences in the occurrence of malformations or anomalies. These NOAEL and LOAEL values from each reliable study for developmental effects in rats are recorded in Table 2-2 and plotted in Figure 2-2. 2.2.2.7 Genotoxic Effects No studies were located regarding genotoxic effects in humans or animals after oral exposure to white phosphorus. Genotoxicity studies are discussed in Section 2.5. 2.2.2.8 Cancer No studies were located regarding cancer in humans or after oral exposure to white phosphorus. In the only chronic duration oral study in animals, no treatment-related histopathological lesions were observed in the lungs or other organs (not otherwise specified) in rats given .1.6 mg/kg/day white phosphorus in the diet for up to 479 days (Fleming et al. 1942). Only six rats per dose group were used. 2.2 DISCUSSION OF HEALTH EFFECTS BY ROUTE OF EXPOSURE
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SP6 IR Resort Anthem#2(ANOTHER) (3/24～3/30 23 30まで)ID：DWZAPYCA STAGE1：☆9 I FIGHT ME STAGE2：☆9 PLASMA STAGE3：☆8 マチコの唄 STAGE4：☆10 Twelfth Style 1 DJ LOVELY 7836 2 DJ JAQK 6790 3 DJ PORTE 6770 4 DJ GONGON 6624 5 DJ ISBA 6585 6 DJ MYNUS 6577 7 DJ 1205 6530 8 DJ TAKARA 6525 9 DJ *369* 6465 10 DJ IDEA-F 6164 11 DJ KUMAAA 6146 12 DJ TETSU 6134 13 DJ MAO 6130 14 DJ 22DUNK 6107 15 DJ BAUYUI 6103 16 DJ KTDC 6102 17 DJ 1059M 6100 18 DJ INS-K 6085 19 DJ VIVIFY 6070 20 DJ TAKI 6059 21 DJ LUV! 6024 22 DJ NAKA.C 6016 23 DJ ASELIA 6004 24 DJ COOPER 5997 25 DJ CRZ 5986 26 DJ N-VANA 5969 27 DJ MAKUW 5906 28 DJ WARAI 5854 29 DJ SANAE 5835 30 DJ *MIO* 5822 31 DJ CHARLE 5810 32 DJ YAYOI 5798 33 DJ GURO** 5798 34 DJ P-KANA 5787 35 DJ ASHLEY 5720 36 DJ PENTUN 5701 37 DJ CINQUE 5695 38 DJ SIHN 5688 39 DJ SMJ 5654 40 DJ DAV-DH 5644 41 DJ TANE 5631 42 DJ PUUTAN 5625 43 DJ HASAMI 5599 44 DJ TEK 5591 45 DJ DEBUZD 5568 46 DJ SASH 5547 47 DJ SYO 5532 48 DJ COLA 5481 49 DJ CAISSO 5388 50 DJ OMAKE 5353 51 DJ GYO29 5338 52 DJ SHI-NO 5322 53 DJ LA-W 5320 54 DJ SUZUME 5308 55 DJ JETS 5301 56 DJ TS# 5301 57 DJ A-SATO 5299 58 DJ TITUS 5294 59 DJ LINUS 5272 60 DJ MAGO 5238 61 DJ MIKOTO 5224 62 DJ YUKI 5219 63 DJ LINE 5210 64 DJ LV.77 5196 65 DJ RATAT 5194 66 DJ TK 5171 67 DJ AUGUST 5165 68 DJ 5193 5118 69 DJ HAIZI 5116 70 DJ HIMMEL 5049 71 DJ AOI 5042 72 DJ WOTA2 5029 73 DJ LASSI. 5023 74 DJ KURO38 5018 75 DJ HRIM 5010 76 DJ TAKA 4967 77 DJ MAITO 4899 78 DJ AMBASA 4895 79 DJ NI-DA* 4777 80 DJ 165CM 4746 81 DJ ZXCV-S 4744 82 DJ ROCHE. 4729 83 DJ ##SU## 4638 84 DJ HEALEN 4593 85 DJ SUGA 4509 86 DJ FGA 4430 87 DJ MARTS 4338 88 DJ NYARIN 4287 89 DJ KRAYZI 4194 90 DJ !!!!!! 3979
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Sorceress 日本名：ソーサレス 略称：Sorc,ソサ,魔女 特徴 攻撃魔法の使い手。炎／雷／氷の三属性を操る。 テレポがあまりに便利。（昨今はどのキャラでもRW Enigmaで飛んでるようですが） hell の敵はほとんど一つ以上の属性に対し無効を持つため、マルチプレイオンリーでなければ、二属性を習得するべき。三属性を習得するのはやめた方がいい。スキルポイントが分散しすぎてどれも弱くなってしまう。 兎に角Lightning Spells のTeleportとStatic Fieldは必修スキル。炎／氷の二属性を選択した場合でもこれだけは取っておくこと。 打たれ弱い代わりに攻撃力は強い。レベルを上げてスキルポイントを稼げば貧弱装備でもなんとかクリアは可能。初心者にも扱いやすいクラス。 Life Mana Stamina Strength Dexterity Vitality Energy Default 40 35 74 10 25 10 35 UP/Lvl 1 2 1 UP/Pts 2 2 1 CastFrame 7 8 9 10 11 12 13 FCR(%) 200 105 63 37 20 9 0 Hit Recovery Frame 4 5 6 7 8 9 10 11 12 13 14 15 FHR(%) 1480 280 142 86 60 42 30 20 14 9 5 0 攻撃手段評価 ★1～★5まで5段階。by名無し熊。 Physical Fire Lightning Cold Poison Magic ★★ ★★★★★ ★★★★★ ★★★★★ ★ ★ ★1 殆ど無い。 ★3 無理すれば。 ★5 メインダメージ源たりえる存在。 スキルポイントや装備をそれ用に注ぎ込んだ場合の評価とする。 スキル Cold Spells Ice Bolt [1] Frozen Armor [1] Frost Nova [6] Ice Blast [6] Shiver Armor [12] Glacial Spike [18] Blizzard [24] Chilling Armor [24] Frozen Orb [30] Cold Mastery [30] Lightning Spells Charged Bolt [1] Telekinesis [6] Static Field [6] Lightning [12] Nova [12] Chain Lightning [18] Teleport [18] Thunder Storm [24] Energy Shield [24] Lightning Mastery [30] Fire Spells Fire Bolt [1] Warmth [1] Inferno [6] Blaze [12] Fire Ball [12] Fire Wall [18] Enchant [18] Meteor [24] Fire Mastery [30] Hydra [30] Tips NPC Introduction 街のNPCには特定のクラスに異なる応対をしてくれる人がいる。 Act入り後初の会話（Talk→Introduction）がクラス専用となるNPCの対応表は以下の通り。 Act1 Act2 Act3 Act4 Act5 Akara Drognan Hratli - Malah
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曲名 作曲者 声ネタ有無 Aegis IT medalion ｆｄｄ ｆｄｄ ALBIDA DJ YOSHITAKA ｆｄｄ ｆｄｄ Answer 星野奏子 ｆｄｄ ｆｄｄ ANTHEM LANDING DJムラサメ ｆｄｄ ｆｄｄ Believe In Me Uraken feat. Calyn Tsukishima ｆｄｄ ｆｄｄ BLUST OF WIND Anemo=Aspel by MLREC. ｆｄｄ ｆｄｄ Bounce Bounce Bounce kors k feat.Kanata Okajima＆楽天斎 ｆｄｄ ｆｄｄ Breaking the ground Art of Fighters ｆｄｄ ｆｄｄ Broken dj TAKA feat.AiMEE ｆｄｄ ｆｄｄ BROKEN EDEN MYTHOLOGIA by MLREC. ｆｄｄ ｆｄｄ Cansei de S NIK PRASTIK DANCEFLOOR ｆｄｄ ｆｄｄ Dances with Snow Fairies Dr.Honda ｆｄｄ ｆｄｄ DAYDREAMER ピンクターボ ｆｄｄ ｆｄｄ Destiny Sword CAPACITY GATE ｆｄｄ ｆｄｄ DM STAR〜関西 energy style〜 NAOKI ｆｄｄ ｆｄｄ ELECTRIC MASSIVE DIVER L.E.D.-G ｆｄｄ ｆｄｄ Energy Drive kevin energy ｆｄｄ ｆｄｄ Eternal Tears ツガル ｆｄｄ ｆｄｄ Everlasting Resort SUPER STAR 満 -MITSURU- ｆｄｄ ｆｄｄ EXTREMA PT.2 Remo-con ｆｄｄ ｆｄｄ Fantasia TЁЯRA ｆｄｄ ｆｄｄ Get Out 壱岐尾彩花 ｆｄｄ ｆｄｄ Golden Palms Dirty Androids ｆｄｄ ｆｄｄ Hardcore Mania DJ Weaver ｆｄｄ ｆｄｄ I FIGHT ME kobo feat.Marsha ｆｄｄ ｆｄｄ Kailua kors k ｆｄｄ ｆｄｄ La Mar seiya-murai feat.David Solanes Venzala ｆｄｄ ｆｄｄ Let The Track Flow Brisk Ham ｆｄｄ ｆｄｄ LIFE SCROLLING HIROSHI WATANABE ｆｄｄ ｆｄｄ Love ♡ km dj TAKA feat.REN ｆｄｄ ｆｄｄ Medicine of love イロハ＆リリナ ｆｄｄ ｆｄｄ Mermaid girl Cream puff ｆｄｄ ｆｄｄ Never Fade Away Sota Fujimori feat. dj MAX STEROID ｆｄｄ ｆｄｄ New Castle Legions Dirty Androids ｆｄｄ ｆｄｄ PARADISE LOST Ryu☆ Vs. L.E.D.-G ｆｄｄ ｆｄｄ passionate fate Ryu☆ ｆｄｄ ｆｄｄ perditus†paradisus conoclasm ｆｄｄ ｆｄｄ Programmed Life kors k ｆｄｄ ｆｄｄ Programmed Sun(xac Antarctic Ocean mix) xac ｆｄｄ ｆｄｄ Raise your hands Keiichi Ueno feat.RIISA ｆｄｄ ｆｄｄ reunion Tatsh ｆｄｄ ｆｄｄ Rock Da House M-Project fw.GUHROOVY ｆｄｄ ｆｄｄ SABER WING TAG ｆｄｄ ｆｄｄ Sakura Reflection Ryu☆ ｆｄｄ ｆｄｄ sakura storm Ryu☆ ｆｄｄ ｆｄｄ Session 9 -Chronicles- PRASTIK DANCEFLOOR ｆｄｄ ｆｄｄ Space Time NISH ｆｄｄ ｆｄｄ SPECIAL SUMMER CAMPAIGN! Lucky Vacuum ｆｄｄ ｆｄｄ Stay my side DJ Yoshitaka feat.RINA ｆｄｄ ｆｄｄ STELLAR WIND L.E.D. ｆｄｄ ｆｄｄ Survival Games VENUS ｆｄｄ ｆｄｄ Take Me Higher Sota Fujimori feat.Sachi ｆｄｄ ｆｄｄ THE BLACK KNIGHT L.E.D. ｆｄｄ ｆｄｄ THE DOOR INTO RAINBOW L.E.D. ｆｄｄ ｆｄｄ THE FALLEN L.E.D.-G ｆｄｄ ｆｄｄ Thunderbolt 雷龍 ｆｄｄ ｆｄｄ Tropical April Risk Junk ｆｄｄ ｆｄｄ Vermillion Mystic Moon ｆｄｄ ｆｄｄ Watch Out Pt.2 DJ Mass MAD Izm* VS. DJ Yoshitaka ｆｄｄ ｆｄｄ WE LOVE SHONAN Studio Bongo Mango feat.Likkle Mai ｆｄｄ ｆｄｄ WISE UP! 木之下慶行 feat.星野奏子 ｆｄｄ ｆｄｄ Wuv U kors k ｆｄｄ ｆｄｄ XANADU OF TWO T.Kakuta With Starving Trancer + 森永真由美 ｆｄｄ ｆｄｄ You Were The One good-cool ft. Brenda Vaughn ｆｄｄ ｆｄｄ ZETA〜素数の世界と超越者〜 Zektbach ｆｄｄ ｆｄｄ おおきなこえで 猫叉Master feat.Sana ｆｄｄ ｆｄｄ かげぬい Ver.BENIBOTAN CULTVOICE by S.S.D.PRODUCTS ｆｄｄ ｆｄｄ ラクエン Feat.Chiharu Chonan -JAKA respect for K.S.K. remix Remixed by JAKAZiD ｆｄｄ ｆｄｄ 中華急行 Ryu☆ ｆｄｄ ｆｄｄ 夕焼け 〜Fading Day〜 JAKAZiD ｆｄｄ ｆｄｄ 恋愛レボリューション21-秋葉工房mix- DJ Command feat. うさ＆ともみん ｆｄｄ ｆｄｄ 旅人リラン XOGO BRYKK ｆｄｄ ｆｄｄ 灼熱Beach Side Bunny DJ Mass MAD Izm* ｆｄｄ ｆｄｄ 黒髪乱れし修羅となりて 村正クオリア ｆｄｄ ｆｄｄ
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Floral Printed Resort Dress onepiece_resort_*_0908.swf bk, wh Shibuya 106 Woman s Fashion Tops 150 Ameba Gold http //info.pico.ameba.net/2010/06/24/new-area-shibuya-japan/
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Oracle Enterprise Service Bus Tips Oracle ESBでのSOAP Headerの設定方法
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ソースコード #include stdafx.h struct ProcessorInfo { unsigned int processors; unsigned int architecture; unsigned int type; unsigned int level; unsigned int revision; bool operator()(void); }; /* XP以降に対応 */ bool ProcessorInfo operator()(void) { this- architecture = 0; this- level = 0; this- processors = 0; this- revision = 0; this- type = 0; HMODULE dll = LoadLibrary(_T( kernel32.dll )); if (dll==NULL) { return false; } typedef void (CALLBACK* GetNativeSystemInfoAPI)(LPSYSTEM_INFO); GetNativeSystemInfoAPI api = (GetNativeSystemInfoAPI) GetProcAddress(dll, GetNativeSystemInfo ); if (api==NULL) { FreeLibrary(dll); return false; } SYSTEM_INFO inf={0}; api(&inf); FreeLibrary(dll); this- architecture = inf.wProcessorArchitecture; this- level = inf.wProcessorLevel; this- processors = inf.dwNumberOfProcessors; this- revision = inf.wProcessorRevision; this- type = inf.dwProcessorType; return true; } int WINAPI WinMain(HINSTANCE,HINSTANCE,LPSTR,int) { ProcessorInfo pinf; if (pinf()) { wchar_t buffer[1000]; swprintf_s(buffer, 1000, L %d Core, Type %d , pinf.processors, pinf.type); MessageBox(NULL, buffer, _T( ), MB_OK); } else { MessageBox(NULL, _T( 取得に失敗しました ), _T( ), MB_OK); } return 0; }