約 3,304,431 件
https://w.atwiki.jp/koitsuda/pages/138.html
会員削除済み (2008/06/05) [Name] [Rating] [Ave] [Win] [Lose] [Game] [BestR] [ID] BarcodeHage 2233 .528 1055 943 2002 四段 128687 (2008/06/02) 将棋倶楽部24ソフト指しはコイツだPart6(248-252) http //game14.2ch.net/test/read.cgi/bgame/1206456861/248-252 248 :名無し名人 :2008/06/01(日) 22 23 42 ID 3LELNUo8 BarcodeHage ボナンザで90%以上一致。 249 :鑑定5号 ◆WidVvGdt3U :2008/06/01(日) 23 56 21 ID JYFxawgf 244-246 いつもお疲れ様です。 248 私も調べてみました。 開始日時:2008/05/31 13 30 33 棋戦:レーティング対局室(早指し) 先手:BarcodeHage (R2238) 後手:(R2142) ▲7六歩1J △3四歩 ▲6六歩1J △8四歩 ▲1六歩1J △1四歩 ▲7八銀1J △6二銀 ▲6八飛1J △5四歩 ▲3八銀1J △4二玉 ▲4八玉1J △3二玉 ▲6七銀1J △7四歩 ▲3九玉1J △5三銀 ▲2八玉1 △5二金右 ▲5八金左1J △8五歩 ▲7七角1J △9四歩 ▲9六歩1J △7三桂 ▲5六歩11 △6四歩 ▲4六歩1 △4四歩 ▲7八飛1 △3三角 ▲7五歩1 △同 歩 ▲5九角1 △6三金 ▲7五飛1 △7四歩 ▲7八飛1 △2二玉 ▲3六歩1 △3二銀 ▲3七桂1 △4三銀 ▲4七金1 △3二金 ▲2六歩1 △4二銀 ▲2五桂1 △2四角 ▲2七銀1 △5三銀 ▲7六銀11 △5一角 ▲7五歩1 △8四飛 ▲7四歩1 △2四歩 ▲7三歩成1 △同 金 ▲3八金11 △2五歩 ▲同 歩1 △3三桂 ▲7五銀11 △8一飛 ▲2四桂2 △3一金 ▲7四銀1 △同 金 ▲同 飛1 △7三歩 ▲7五飛1 △2五桂 ▲6三金1 △4二銀 ▲7二金1 △8三飛 ▲2五飛1 △2三歩 ▲6一金2 △2四歩 ▲同 飛1 △2三銀 ▲2六飛1 △8一飛 ▲5一金1 △同 飛 ▲2四歩1 △1二銀 ▲1五歩1 △同 歩 ▲1三歩1 △同 玉 ▲1五香1 △1四歩 ▲同 香1 △同 玉 ▲2三角1 △同 銀 ▲1五歩1 △同 玉 ▲1六銀1 まで103手で先手の勝ち ボナンザv2.1の最善・次善手との一致率 48/52=92.3%(8053113)【認5】。 250 :鑑定5号 ◆WidVvGdt3U :2008/06/01(日) 23 57 12 ID JYFxawgf 開始日時:08/06/01 18 22 17 棋戦:レーティング対局室(早指し) 先手:(R2199) 後手:BarcodeHage (R2238) ▲7六歩 △3四歩1J ▲6六歩 △8四歩1J ▲6八飛 △8五歩1J ▲7七角 △6二銀1J ▲4八玉 △5四歩1J ▲3八銀 △4二玉1J ▲3九玉 △3二玉1J ▲1六歩 △5三銀1J ▲7八銀 △3三角2 ▲5八金左 △2二玉1J ▲4六歩 △1二香1 ▲3六歩 △1一玉1 ▲3七桂 △4四歩1 ▲6七銀 △2二銀1 ▲2六歩 △3一金1 ▲6五歩 △5一金2 ▲2八玉 △4一金右1 ▲2七銀 △7四歩1 ▲3八金 △7二飛1 ▲6六銀 △5一角1 ▲5六歩 △3二金右11 ▲4七金左 △8二飛2 ▲1五歩 △8四角1 ▲9六歩 △9四歩1 ▲5七銀 △4三金11 ▲5五歩 △同 歩1 ▲同 角 △7三桂1 ▲5六銀 △5四銀1 ▲8八角 △5五歩1 ▲同 銀 △同 銀1 ▲同 角 △8六歩11 ▲同 歩 △5四金1 ▲8八角 △6五桂1 ▲1四歩 △同 歩1 ▲1三歩 △同 香1 ▲2五桂 △5七桂成1 ▲同 金 △同角成1 ▲6三飛成 △5三金打1 ▲6一龍 △8六飛1 ▲1三桂成 △4六馬1 ▲3七銀 △1三馬1 ▲2五桂 △2四馬1 ▲1三歩 △同 桂1 ▲同桂成 △同 馬1 ▲2五桂 △2四馬1 ▲1三歩 △2一玉1 ▲3三香 △同 銀1 ▲1二歩成 △3二玉1 ▲9七角 △8九飛成1 ▲3三桂成 △同 馬1 ▲5五歩 △6四金寄1 ▲同 角 △同 金1 ▲同 龍 △4五桂1 ▲6二龍 △4二香1 ▲5四銀 △3七桂成1 ▲同 玉 △4五桂1 ▲4七玉 △5七桂成1 ▲4六玉 △4五銀1 ▲5七玉 △5六銀打1 ▲5八玉 △4六桂1 ▲4八玉 △3八桂成1 ▲同 玉 △4七金1 ▲2八玉 △3七角1 ▲1七玉 △1九龍1 ▲1八金 △2八角成1 まで130手で後手の勝ち ボナンザv2.1最善・次善手との一致率 62/65=95.4%(806014)【認5】。 251 :初代624 ◆1zrHcb/LWY :2008/06/02(月) 22 08 59 ID yON9P4wZ 244-246 249-250 乙です 開始日時:2008/05/17 12 09 12 棋戦:レーティング対局室(早指し) 後手:BarcodeHage ▲76歩 △34歩 ▲66歩 △33角 ▲78飛 △22飛 ▲36歩 △24歩 ▲38銀 △25歩 ▲37銀 △42銀 ▲38金 △72銀 ▲75歩 △54歩 ▲58金 △62玉 ▲48玉 △71玉 ▲68銀 △94歩 ▲67銀 △14歩 ▲46歩 △53銀 ▲39玉 △82玉 ▲96歩 △52金左▲74歩 △同歩 ▲同飛 △73歩 ▲78飛 △64歩 ▲76銀 △63金 ▲48金左△44銀 ▲65歩 △同歩 ▲同銀 △64歩 ▲56銀 △55歩 ▲47銀 △74歩 ▲28玉 △15歩 ▲66角 △73銀 ▲86歩 △84銀 ▲97桂 △65歩 ▲77角 △75銀 ▲59角 △72金 ▲95歩 △同歩 ▲85桂 △84歩 ▲92歩 △同香 ▲93歩 △同桂 ▲同桂成△同香 ▲87桂 △64銀 ▲95香 △同香 ▲同桂 △94歩 ▲85歩 △95歩 ▲84歩 △83歩 ▲同歩成△同金 ▲85歩 △72桂 ▲45香 △26歩 ▲同銀 △45銀 ▲同歩 △23香 ▲95角 △26香 ▲同歩 △94歩 ▲86角 △75銀打 ▲59角 △56歩 ▲同歩 △73香 ▲37金左△66歩 ▲68歩 △86歩 ▲77飛 △65桂 ▲79飛 △57桂成▲44歩 △47成桂▲同金寄△44角 ▲46香 △26角 ▲同角 △同飛 ▲27歩 △22飛 ▲55桂 △同銀 ▲同歩 △58銀 ▲48金引△26歩 ▲同歩 △67歩成▲41角 △62金 ▲31銀 △26飛 ▲27歩 △36飛 ▲47銀 △同銀不成▲同金直△38飛成 ▲同玉 △35桂 ▲22飛 △47桂成▲同玉 △65角 ▲37玉 △38金 まで144手で後手の勝ち 後手:(54,61)/66=(81.8,92.4)% Bonanza v2.1の最善・次善手との一致率61/66=92.4%(805175)【認7】 252 :初代624 ◆1zrHcb/LWY :2008/06/02(月) 22 35 43 ID yON9P4wZ 開始日時:2008/05/19 21 45 53 先手:BarcodeHage ▲76歩 △34歩 ▲16歩 △44歩 ▲26歩 △42飛 ▲68玉 △72銀 ▲25歩 △33角 ▲78玉 △32銀 ▲77角 △62玉 ▲88玉 △71玉 ▲48銀 △52金左▲98香 △45歩 ▲33角成△同銀 ▲99玉 △46歩 ▲同歩 △同飛 ▲88銀 △42飛 ▲79金 △46歩 ▲65角 △54角 ▲同角 △同歩 ▲86角 △82玉 ▲42角成△同金 ▲58金 △94歩 ▲68金寄△95歩 ▲56歩 △47歩成▲同銀 △39角 ▲58飛 △84角成 ▲31飛 △32角 ▲55歩 △同歩 ▲同飛 △41金 ▲同飛成△同角 ▲51金 △52歩 ▲61金 △同銀 ▲51金 △62金 ▲41金 △44飛 ▲31金 △47飛成▲21金 △41龍 ▲11金 △同龍 ▲86香 △74馬 ▲75桂 △72銀 ▲45飛 △44銀打▲49飛 △41龍 ▲54歩 △84歩 ▲56角 △65金 ▲同角 △同馬 ▲84香 △71玉 ▲82金 △61玉 ▲91金 △38角 ▲59飛 △76馬 ▲78香 △75馬 ▲同香 △83歩 ▲45歩 △84歩 ▲44歩 △同銀 ▲45歩 △55香 ▲39飛 △65角成 ▲44歩 △54馬 ▲43銀 △44馬 ▲42銀打△21龍 ▲49飛 △48歩 ▲同飛 △45歩 ▲65角 △12龍 ▲53歩 △同歩 ▲81金 △同銀 ▲74歩 △同歩 ▲同香 △73歩 ▲36桂 △35馬 ▲73香成△同金 ▲74歩 △72金 ▲24桂 △同歩 ▲36歩 △44馬 ▲46歩 △64歩 ▲45歩 △26馬 ▲28飛 △36馬 ▲24歩 △26歩 ▲38飛 △45馬 ▲34銀不成△63馬▲43角成△71玉 ▲23銀不成△11龍▲53銀成△74馬 ▲32飛成△82玉 ▲54馬 △51香 ▲52歩 △41金 ▲43龍 △42歩 ▲同成銀△同金 ▲同龍 △52馬 ▲33龍 △43歩 ▲64馬 △73歩 ▲22銀成△76桂 ▲11成銀△68桂成▲同金 △76桂 ▲78金 △69銀 ▲79金打△58香成▲94桂 △71玉 ▲44歩 △78銀成▲同金 △68成香 ▲同金 △同桂成▲79香 △同成桂▲同銀 △76香 ▲77歩 △69金 ▲88銀打△68金打▲同銀 △同金 ▲93桂 △78金打▲81桂成△同玉 ▲82銀 △同金 ▲91金 △同玉 ▲93飛 まで205手で先手の勝ち 先手:(87,97)/99=(87.9,98.0)% Bonanza v2.1の最善・次善手との一致率:97/99=98%(805192)【認7】
https://w.atwiki.jp/sutona/pages/120.html
■先行SUT○NA(2009年10月03日) 15 08 (SUTONA) SUTONAさん独走状態っ!全ての人の先を行く者なんですっ!!!200年後を先取りしてるんですっ!!!111相手は死ぬんですっ!!!!1!!!!1111 15 08 (ranran_) どういう指してるのか気になるわw 15 08 (ranran_) これ打つの時間かかったのかな 15 09 (e) 15 08 (SUTONA) SUTONAさん独走状態っ!全ての人の先を行く者なんですっ!!!200年後を先取りしてるんですっ!!!111←3の倍数相手は死ぬんですっ!!!!1!!!!1111←2進数で言うところの15=3の倍数 15 10 (e) SUT○NAさん=先行者と 15 10 (e) 覚えた。 15 10 (e) 股間からビーム出るのか… 15 10 (e) はっ!? 15 10 (SUTONA) 先行者いやぁぁぁぁあああああああああああああああああああああああああああああああああああああああ!!!!!!!!!!11111111111 15 10 (e) グングニルってそれかああああああああ 15 10 (SUTONA) ちーーーgはーーーーうーーーーー!!!!!!!!!!!!!!!!!!1111111111111 15 10 (SUTONA) が! 15 11 (e) どうやって間違うのそれ… 15 11 (Akurosu_Arufu) 訂正をしようとしても結局誤字るSUTONAさん、 15 11 (Akurosu_Arufu) かわいいな 15 11 (e) ホットコーヒーが鼻から出るわ(´ω`) 15 12 (ranran_) 君のハートに股間からグングニル☆今夜は僕と不夜城レッド☆ 15 12 (SUTONA) 嫌ぁぁああああああああああああああああああぁぁぁああああああああああああああああああああああああああああ!!!!!!!!!!1111111111 15 12 (e) http //www6.plala.or.jp/private-hp/samuraidamasii/kikaku/kahou/kahou73.gif 15 12 (e) ↑グングニル 15 13 (ranran_) もう、SUTONAさんそのものだね 15 13 (Akurosu_Arufu) \すげぇ/ 15 13 (SUTONA) 先行者は嫌・・・・・・・・・ 15 13 (ranran_) 流石すぎて言葉もないわw 15 15 (ranran_) その股間からグングニルで今度は誰をおとす気なんだろう(わくわく 15 16 (SUTONA) グングニルは槍っ!!!!!!!!!!!111111111111111 15 16 (e) ↑3の倍数 15 16 (SUTONA) ・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・ 15 16 (suragu) ○槍 15 16 (suragu) さて○には何が入るのだろうね 15 17 (SUTONA) 神! 15 17 (ranran_) 無理槍 15 17 (e) 中華槍 15 18 (Akurosu_Arufu) 神槍でもSUTONAさんの場合卑猥な表現になると思うよ 15 18 (SUTONA) ・・・・・・・ 15 19 (suragu) てかさ 15 19 (suragu) 15 15 (SUTONA) ・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・・ 15 19 (suragu) って迷う時点で変な想像してたんじゃないかね? 15 19 (SUTONA) !* 15 19 (SUTONA) !? 15 19 (e) アスタリスクときたか・・・ 15 20 (SUTONA) なんで・・・・・・・ 15 20 (suragu) *+槍=? 15 20 (e) こっちが聞きたいわ(´ω`) 15 20 (suragu) *+槍=アッー 15 20 (suragu) と言う公式が成り立ちます 15 20 (e) あぶない 15 21 (e) コーヒー口に含もうとして↑のみた 15 21 (e) もう数秒遅かったらディスプレイ■大三字だよ 15 21 (suragu) w 15 21 (SUTONA) にゃぁぁぁあああああああああああああああぁぁあっぁぁぁああああああああああああああああああ!!!!!!!!!!1111111111111 15 22 (ranran_) 卑猥すぎるw 15 23 (e) SUT○NAさんはホイホイついてくるノンケが大好きです。 15 23 (SUTONA) !??!?!?!?!?!!?!?!?!?!??!?!?!!?? 休日の昼間から…いいぞ、もっとやれ。
https://w.atwiki.jp/satoschi/pages/302.html
エドイド諸語 Niger-Congo Atlantic-Congo【alv】 Volta-Congo Benue-Congo Edoid languages Delta — Degema【deg】—デゲマ語 Engenni【enn】— Epie【epi】—エピエ語 North-Central —北中部諸語 Edo-Esan-Ora —エド=エサン=オラ語 Edo【bin】—エド語 Emai-Iuleha-Ora【ema】—エマイ=イウレハ=オラ語 Ibilo【ibi】→ Retired. Merge into Okpamheri【opa】 Esan【ish】—エサン語 Ghotuo-Uneme-Yekhee —ゴトゥオ=ウネメ=イェケエ語 Ghotuo【aaa】—ゴトゥオ語 Ivbie North-Okpela-Arhe【atg】— Enwan【env】— Yekhee【ets】—イェケエ語 Igwe【igw】—イグウェ語 Ikpeshi【ikp】— Ososo【oso】— Sasaru【sxs】— Uneme【une】—ウネメ語 Ihievbe【ihi】— Uokha【uok】— Northwestern —北西部諸語 Aduge【adu】— Osse — Ehueun【ehu】—エフェウン語 Iyayu【iya】—イヤユ語 Uhami【uha】—ウハミ語 Ukue【uku】—ウクウェ語 Southern —南部諸語 Akuku【ayk】—アクク語 Idesa【ids】—イデサ語 Okpe【okx】—オクペ語 Oloma【olm】—オロマ語 Okpamheri【opa】—オクパメリ語 Southwestern—南西部諸語 Eruwa【erh】—エルワ語 Uvbie【evh】—ウビエ語 Isoko【iso】—イソコ語 Okpe【oke】—オクペ語 Urhobo【urh】—ウルホボ語
https://w.atwiki.jp/stgl2014/pages/254.html
自己紹介 研究済んでるのに、お金がなくて買えないのがいぱーいになってきました>< 所有車両リスト Tier 軽戦車 中戦車 重戦車 駆逐戦車 自走砲 1 2 MKA T-45 T7 Combat Car T-26G FT ARM 35 L-60 Pz.kpfw.II Ausf.D Tetrarch Type97 Te-ke Cruiser Mk.III Light Mk.VIC TKS z n.k.m.20mm T82 3 Pz.kpfw. I Ausf.C Pz.Kpfw. T 15 T-116 T-29 Cruiser Mk.II 43 M.Toldi III BT-7 artillery LTP MTLS-1G14 Type 2597 Chi-Ha 10TP Pz.kpfw.T-15 LT vz.38 T-29 M15/42 FCM 36 Pak40 4 T-50 Pz.Kpfw.II Luchs Pz.kpfw III Matilda T-34 with L-11 P26/40 Hetzer SU-76G FT Pz.Stl. IC 5 VK 16.02 Leopard AMX ELC bis M24 Chaffee M4 Sherman Pz.Kpfw.IV Ausf.H Valiant M4A2E4 Sherman DS PZInz P.43 Type T-34 Pz.Kpfw.III/IV Type3 Chi-Nu Kai 25TP KSUST II Strv m/42 T-34 Sherman III KV-1 O-I Experimental BDR G1 B Churchill I T67 StuG IV M10 RBFM S35 CA StuG3 SU-85 Pz.Sfl.IVc AT2 M41 HMC Grille 6 MT25 Crusader T37 59-16 T-50-2 A46 M24E2 Super Chaffee AMX 12 t T-34-85 Cromwell B.U.G.I. Strv 74 A-43 Skoda T 25 T-34-85M Sherman Firefly 40TP Habicha Strv m/42-57 Alt A.2 Type 58 P.43 bis Type4 Chi-To M4A3E8 VK30.01P M4A3E2 Sherman J KV-2 Heavy Tank No. VI O-I churchill VII KV-85 ARL44 T-150 KV-1S SU-100 M18 Hellcat Dicker Max SU-100Y WZ-131G FT AT8 M36 Jackson Jagdpanzer IV ARL V39 Nashorn FV304 AMX 13 F3 AM M44 Hummel 7 LTG WZ-131 T71 DA AMX13 75 Comet Leo T-34-1 P.43 ter CS-44 Type 5 Chi-Ri Konstrukta T-34/100 T-43 A-44 KV-13 IS FV201(A45) KV-3 O-Ni Black Prince IS-2M 45TP Habicha T29 IS-2 Tiger(P) Tiger I AMX M4 mle.45 SU-100M1 M56 Scorpion T25/2 Super Hellcat SU-152 T28 Concept Sturer.Emil AT7 JagdPanther AMX AC mle. 46 T25AT S-51 Crusader 5.5-in. SP G.W.Panther 8 Panhard AML Lynx 6x6 M41 Walker Bulldog ELC EVEN 90 Bat-Chatillon 12 t T69 TVP VTU Koncept T-44 Centurion Mk. I P.44 Pantera STA-1 T-34-2 IS-2-II AMX M4 mle.49 Liberte Emil I KV-4 110 AMX 50 100 O-HO 53TP Markowskiego Tiger II IS-3 Caernavon T-32 VK45.02(P)Ausf.A Rhm Borsig T28 Prototype AT 15 8.8cmPak43 Jagdtiger ISU-152 AMX AC mle.48 Rheinmetall SkorpionG Charioteer JagdPanther II T28 Ferdinand M40/43 SU-14-2 G.W.Tiger(P) FV207 9 T49 AMX13 90 Panhard EBR 90 T-54 Prototipo Standard B Bat-Chatillon 25 t AP UDES 16 E75 Object 257 Emil ll M103 T-10(IS-8) Type 4 Heavy VK45.02(P)Ausf.B Waffentrager auf Pz.IV K-91-PT T30 AMX 50 Toch Object 704 Jagdtiger T95 Tortoise Strv 103-0 M53/M55 bat-chatillon 155 55 212A 10 FV215b Maus E100 T110E4 AMX 50 Foch B Bat-chatillon 155 58
https://w.atwiki.jp/normter/
経済産業省へのメールテンプレート RMT抗議運動/Protest against RMT 現在、尻鯖ではサービス提供外(中国、台湾、ベトナムなど)からのギル生産者が多くいます。これらのキャラクターは、固定PTでレベルを一定レベルまで上げ、それ以降はギル生産を行い続けます。ギル生産の手段は、栽培、採掘、HNM、NM、合成、傭兵などあるみたいです。 There is lot of gil farmer from China, Taiwan and Vietnam on Quetzalcoatl Server. Those characters are leveling in static party and when they got reach to certain level they start farming gil for 24 hours. (Dead?)Group 1 詳細(Detail) BAS6 BLM75/RDM35 HUM BeckHamk (LS DragonWarriorr) BAS6 BLM75/RDM35 TAR Buhui (LS DragonWarriorr) BAS6 BLM75/RDM34 TAR Dauday (LS DragonWarriorr) BAS6 WHM75/BLM35 TAR Emex (LS DragonWarriorr) BAS6 PLD75/WAR34 ELV Feier (LS DragonWarriorr) BAS6 BLM75/RDM33 MIT Kissf (LS DragonWarriorr) BAS6 PLD75/WAR35 HUM Ronaldos (LS DragonWarriorr) BAS6 BLM75/WHM33 TAR Thecry (LS DragonWarriorr) BAS6 BRD75/WHM36 HUM Kellys (LS DragonWarriorr) BAS6 WHM75/BLM35 TAR Hhllyk (LS DragonWarriorr) BAS6 BLM75/WHM35 TAR Killyn (LS DragonWarriorr) BAS6 BLM75/RDM35 TAR Rockcoat (LS DragonWarriorr) Group 2 詳細(Detail) SND1 WHM74/BLM37 TAR Angeli SND1 BLM74/RDM37 TAR Biririg SND1 BLM74/RDM37 TAR Rattys SND1 BLM74/RDM37 HUM Ubyt SND1 BLM74/RDM37 HUM Untat SND1 BLM74/RDM37 TAR Kemen Group 3 詳細(Detail) SND1 BLM3 TAR Bigfish SND1 WAR4 HUM Lemar SND1 BLM6 TAR Ohayo SND1 BLM5 TAR Moln SND1 BLM4 HUM Dreamscometrue SND1 BLM4 TAR Overbe Group 4 詳細(Detail) SND10 RDM75/BLM37 TAR Annaa (LS Amiability) PL SND6 PLD75/WAR37 HUM Ahtao SND6 BLM75/RDM37 TAR Ahbu SND6 RNG75/NIN37 HUM Feir (LS Amiability) SND6 MNK75/WAR37 ELV Mede (LS BlueHNM) SND6 WHM75/BLM37 TAR Xinzi SND6 RDM75/WHM37 HUM Yuer Group 5 詳細(Detail) Evesky TAR Wljsky HUM Shalf TAR Sckugli HUM Yitou TAR SND2 WHM33/BLM16 TAR Sqsm Group 6 詳細(Detail) SND6 BLM75/WHM37 TAR Mamuamua SND6 THF75/NIN37 MIT Pahoho SND6 NIN75/WAR37 ELV Febabont SND6 RDM75/BLM37 HUM Fotinn (LS fortinn) SND6 WHM75/BLM37 TAR Duaduadudu SND6 THF75/NIN37 MIT Tunnune Group 7 (PL by Krebs) 詳細(Detail) BAS10 THF75/NIN37 MIT Wukyu BAS10 BLM75/RDM37 TAR Minra BAS10 THF75/NIN37 MIT Cehih BAS10 Rnahye BAS10 Jegovya BAS10 NIN75/WAR37 ELV Uvweie Group 8 詳細(Detail) SND9 WHM74/BLM37 TAR Feya (LS Amiability) PL SND6 BLM75/RDM37 TAR Bupabupa SND6 BLM75/RDM37 TAR Feisei SND6 BLM75/RDM37 HUM Lysha (LS Fantasyangel) SND6 BLM75/RDM37 TAR Mihuh (LS Fantasyangel) SND6 BLM75/RDM37 TAR Vitehite SND6 BLM75/RDM37 TAR Yaoyao Group 9 詳細(Detail) BAS10 MNK75/NIN37 ELV Uaeevil BAS10 BLM75/WHM37 TAR Feliheli BAS10 NIN75/WAR35 ELV Dicond BAS10 BRD75/WHM37 HUM Ahy BAS10 WHM75/BLM37 TAR Nodondon BAS10 THF75/NIN37 MIT Rore Group 10 詳細(Detail) SND10 RDM75/BLM37 TAR Annaa (LS Fantasyangel) SND9 WHM74/BLM37 TAR Feya (LS Fantasyangel) Heym (LS Fantasyangel) Laja (LS Fantasyangel) SND9 THF75/WAR5 TAR Notenate (LS Fantasyangel) SND9 BLM74/WHM37 TAR Repkaka (LS Fantasyangel) Group 11 詳細(Detail) Standby (LS HNMLINKS) PL Baishill Jimese Jinlaik Miaux Twinslele Wahaha Group 12 詳細(Detail) SND6 BLM69/RDM34 TAR Deathmoon Evilqueen SND6 NIN69/WAR34 ELV Fanking (LS WGleague) SND6 THF69/NIN34 MIT Goldskiner SND6 RDM69/WHM34 HUM Joinlin SND6 WHM69/BLM34 TAR Snowshow (Dead?)Group 13 詳細(Detail) SND1 NIN24/WAR12 HUM Asadssadasdads SND1 BRD25/WHM11 TAR Ddjru SND1 WAR25/MNK12 GAL Sohou Kllrou Dssdfsdfsdfsdf SND1 NIN27/THF13 MIT Quder Group 14 詳細(Detail) BAS1 BLM21 TAR Bantoluto BAS1 WAR21 MIT Bynoyoeh BAS1 RDM21 HUM Daphyll BAS1 WHN21 TAR Jaryularyu BAS1 WAR21 MIT Neheh BAS1 BLM21 TAR Hawehyaweh BAS1 BLM21 TAR Gedottogeto BAS1 WAR20 GAL Sailingtide BAS1 WHM19 HUM Clairilda BAS1 THF20 MIT Fayeeh BAS1 WHM19 TAR Fyipsiyafyipya BAS1 BLM19 TAR Okohtsokoh Group 15 詳細(Detail) BAS1 BLM14 TAR Bajenjjokenjo BAS1 WAR15 GAL Euphori BAS1 THF15 MIT Heyaoya BAS1 WHM15 HUM Nathania BAS1 WHM15 TAR Rakehtkyokeht BAS1 BLM14 TAR Wanoyano BAS1 WAR15 GAL Waterless BAS1 WHM16 TAR Nehuchihu BAS1 BLM16 TAR Myuppyepye BAS1 RDM17 HUM Afanasiev BAS1 WAR16 ELV Alcaurie BAS1 BLM15 TAR Nerruperru Group 16 詳細(Detail) BAS1 WHM20 TAR Bapinonyaapino BAS1 BLM018 TAR Bohigyunhi BAS1 WHM19 HUM Fedila BAS1 WHM19 TAR Rehpyee BAS1 BLM19 TAR Ryaffugafu BAS1 WAR19 GAL Unassailablefin BAS1 BLM20 TAR Chigosigo BAS1 RDM20 HUM Midzuwe BAS1 WAR20 GAL Stupidrainbow BAS1 MNK20 GAL Unflinchingjaw BAS1 WHM19 TAR Postcaste BAS1 BLM20 TAR Rudenunde Group 17 詳細(Detail) BAS1 RDM22 HUM Brandie BAS1 BLM21 TAR Pehehhnohyueho BAS1 BLM21 TAR Berihdabepida BAS1 WAR21 GAL Sinuoussparrow BAS1 WAR21 ELV Elmimapise BAS1 WHM21 TAR Jesahmbisahm Group 18 詳細(Detail) TAR Annaarnold TAR Bertiecamp TAR Charleschild TAR Eiizabeth TAR Dickens TAR Fastfelix HUM Flanna (Dead?)Group 19 詳細(Detail) SND6 BLM73/WHM36 TAR Blackboard SND6 NIN75/WAR37 ELV Ninjaninja SND6 PLD74/WAR37 ELV Kood SND6 THF74/WAR07 ELV Loveo SND6 RDM72/BST6 TAR Hoohoo (LS KINGKONG) Severe (Dead?)Group 20 詳細(Detail) BAS6 RDM75/DRK37 HUM Areek Brawlking BAS6 BLM75/WHM37 TAR Darkangelz BAS6 NIN75/WAR37 ELV Endurpains BAS6 WHM75/SMN37 TAR Goddessl (LS DownFall) Slide Group 21 詳細(Detail) SND8 RDM75/BLM37 TAR Arise (PL) ELV Areivl ELV Dmug ELV Kanay (LS SpongDaggers) TAR Valous TAR Monarchdragon (LS SpongDaggers) TAR Forud (LS SpongDaggers) Group 22 詳細(Detail) BAS6 BLM75/WHM37 TAR Genders TAR Pressed MIT Redaudy (LS WigSplitter) ELV Chossese BAS6 THF75/NIN37 HUM Goodlove (LS WigSplitter) BAS6 NIN75/WAR37 ELV Randomn (LS WigSplitter) Group 23 詳細(Detail) BAS1 PLD75/WAR37 ELV Keybads BAS1 NIN75/WAR37 ELV Genlear HUM Hosetow BAS6 BLM75/RDM37 TAR Coolboys TAR Coolgirls BAS6 BLM75/WHM37 TAR Lettewhm BAS6 WHM75/BLM37 TAR Anerte (LS WigSplitter) BAS6 MNK75/WAR37 GAL Recomm (LS WigSplitter) BAS6 WHM75/BLM37 TAR Rockgirls (LS WigSplitter) Group 24 詳細(Detail) SND2 WAR23/MNK05 HUM Coooooooool (LS WePokeSeaHorrors) SND1 BLM23 TAR Kissmeme SND2 WAR23/MNK06 ELV Xingmissyouning (LS WePokeSeaHorrors) SND2 WHM23/BLM01 HUM Bluestrr (LS PandeFariMageBender) SND2 WHM22 TAR Goodbaby (LS WonFourAll) SND2 THF23 MIT Kingta Group 25 詳細(Detail) SND2 WHM62/BLM30 TAR Fltba SND2 BLM62/WHM31 TAR Ooooooooopa SND2 NIN63/WAR31 MIT Soyouta SND2 WHF63/NIN30 MIT Suiyyter SND2 MNK62/WAR18 GAL Tataluli SND2 RDM62/WHM30 TAR Yado Group 26 詳細(Detail) SND1 WAR23 ELV Deanbill SND1 BLM24/WHM01 HUM Hellen SND1 THF24/WAR05 MIT Kittycats SND1 BLM24 TAR Michaeles SND1 WAR24 ELV Peryonjon (LS TeaPartyMasters) SND1 WHM23 TAR Wendy Group 27 詳細(Detail) SND1 PLD27/WAR13 ELV Blackeagle (LS WonForAll) SND1 THF26/NIN13 MIT Invisible (LS BadBoy) SND1 BLM27/WHM13 TAR Mataiiu SND1 DRK27/WAR13 ELV Galanz SND1 WHM25/BLM02 TAR Litter SND1 RDM26/BLM13 ELV Kuangta SND1 RDM10 HUM Marrykiss (Gone?) Group 28 詳細(Detail) SND1 WAR10 ELV Jamesho SND1 RDM10 TAR Anniejin SND1 THF10 ELV Ashleyy SND1 MNK11 ELV Phoehe SND1 BLM11 TAR Tomny SND1 WHM11 TAR Uailin Group 29 詳細(Detail) TAR Ailsa TAR Moyila SND1 WAR11 ELV Songy SND1 WAR11 ELV Dunan SND1 WHM11 ELV Beautifu SND1 WHM10 TAR Roliane Group 30 詳細(Detail) BAS1 WHM15 TAR Ailisi BAS1 THF17 MIT Efvb BAS1 BLM16 TAR Rewed BAS1 RDM16 HUM Scuyh BAS1 WAR16 ELV Sevenleo BAS1 WAR16 ELV Yjmn Group 31 詳細(Detail) SND2 WAR13 HUM Avenge SND2 BLM13 TAR Beatingdragon SND2 WAR13 ELV Hurrcane SND2 WHM13 TAR Mermaider SND1 RDM13 HUM Moonlight SND1 THF13 MIT Vnicorn Group 32 詳細(Detail) SND1 WHM25 TAR Raull SND1 TAR Rubn SND1 GAL Gutti SND1 GAL Beckhamm SND1 THF26 MIT Figoo SND1 HUM Rcarlos Group 33 詳細(Detail) SND1 RDM25 HUM Casillas SND1 THF26 MIT Cambiasso SND1 MNK26 GAL Incrediblefight SND1 WAR28/MNK01 GAL Ronaldoo SND1 BLM29/MNK01 TAR Zidanea SND1 WHM28/BLM01 TAR Solarii Group 34 詳細(Detail) SND1 RDM58/BLM29 HUM Amigogo (LS SecondStringHero) SND1 BLM58/WHM29 TAR Ayaaa (LS ThoseOtherGuys) SND1 NIN58/WAR29 HUM Kidomaru (LS TheRoadLessTravel...) SND1 WHM58/BLM29 TAR Koyukii SND1 MNK58/WAR29 GAL Maitogai SND1 THF58/NIN29 MIT Sakurarara Group 35 詳細(Detail) Ubyt Sukeiky Dempsey SND2 BLM24/WHM12 HUM Abigaley SND3 NIN24/WAR10 ELV Alwan SND1 BLM24/WHM12 TAR Angeli SND5 THF63/NIN31 HUM Amro SND3 WAR64/NIN32 HUM Kaufmann Group 36 詳細(Detail) BAS1 BLM08 HUM Alviua BAS1 WAR11 ELV Beaudant BAS1 THF10 ELV Bilotie BAS1 WAR10 MIT Hervih BAS1 WAR12 ELV Demolaman BAS1 WHM11 TAR Rofore Group 37 詳細(Detail) SND1 BLM17 TAR Anata SND1 BLM17 TAR Melodo SND1 THF17 MIT Striky SND1 BLM17 TAR Uniom SND1 WAR17 ELV Mcbwl SND1 WHM17 HUM Visala Group 38 詳細(Detail) BAS1 WAR17 ELV Redpair BAS1 THF19 HUM Bigfacecat BAS1 WHM18 TAR Tiwcr BAS1 BLM18 TAR Smaldevil BAS1 RDM18 HUM Redrem BAS1 WAR17 GAL Maletyrant Group 39 詳細(Detail) BAS1 WHM09 TAR Barrryssc BAS1 WHF11 MIT Daoqtalvv BAS1 BLM10 TAR Heilfoyssc BAS1 WHM10 HUM Hongxayssc BAS1 WAR11 ELV Rtthkafgfg BAS1 WAR12 ELV Rtvynuftj Group 40 詳細(Detail) SND1 RDM09 HUM Daiwei SND1 WHM09 TAR Dapipi SND1 WAR10 ELV Dargke SND1 THF10 MIT Devilgirl SND1 WAR10 ELV Laodu SND1 BLM08 TAR Qiuqiu Group 41 詳細(Detail) SND1 WAR14 GAL Firewar SND1 WAR14 GAL Freerun SND1 WHM14 TAR Kissyeah SND1 BLM14 TAR Mzeast SND1 BLM14 TAR Smitte SND1 RDM14 TAR Vivianr SND1 PLD37/WAR18 GAL Vunt Group 42 詳細(Detail) SND1 BLM14 TAR Babiwa SND1 WHM14 HUM Bluegirl SND1 THF14 ELV Keypadd SND1 BLM14 TAR Laward SND1 WAR14 GAL Salahio SND1 MIT Shanaxi (LS WonForAll) Group 43 詳細(Detail) (JunnyがPL) SND1 WAR10 MIT Alic (LS WhiteCat) SND1 THF10 MIT Hpmp (LS WhiteCat) SND1 WHM10 MIT Jodi (LS WhiteCat) SND1 RDM10 MIT Lyna (LS WhiteCat) SND1 WAR10 MIT Vinn (LS WhiteCat) SND1 THF10 MIT Yota (LS WhiteCat) Group 44 詳細(Detail) SND1 WHM13 TAR Ryuhefooluhfoo SND1 WAR14 HUM Aurele SND1 RDM14 HUM Biantou SND1 BLM14 TAR Modagida SND1 THF15 MIT Bohfisoo SND1 WAR15 ELV Ricien Group 45 詳細(Detail) BAS1 HUM Ailisha BAS1 ELV Paman BAS1 TAR Sahala BAS1 MIT Yidamida BAS1 TAR Sunson BAS1 WAR17 ELV Pabaut Group 46 詳細(Detail) SND1 TAR Whrich SND1 ELV Lvoro SND1 ELV Kialye SND1 WAR12 ELV Andrewken SND1 BLM12 TAR Anglesmileyangl SND1 WAR20/THF01 ELV Belll SND1 BLM21/RDM10 HUM Morieadan SND1 WAR21/MNK10 HUM Suryael Group 47 詳細(Detail) BAS1 WAR18 ELV Bableu BAS1 BLM17 TAR Chadey BAS1 RDM18 HUM Edette BAS1 WHM18 TAR Gedode BAS1 RDM18 TAR Hajeso BAS1 WHM18 TAR Hunose BAS1 BLM17 TAR Manpiru BAS1 RDM18 TAR Tomotan BAS1 RDM14 TAR Banis BAS1 WHM14 HUM Dragy BAS1 THF14 MIT Larita BAS1 RDM14 TAR Nishiku BAS1 RDM14 TAR Runniw BAS1 RDM14 TAR Seriresi BAS1 BLM14 TAR Gerya BAS1 BLM14 TAR Sabila BAS1 BLM18 TAR Pashor BAS1 WAR16 GAL Undaunted Group 48 詳細(Detail) SND1 THF15 MIT Botzsyl SND1 WHM15 TAR Gootreem SND1 WAR15 ELV Michaol SND1 WAR15 GAL Pandomee SND1 WHM15 TAR Yimikoo SND1 WHM16 TAR Nokeey Group 49 詳細(Detail) SND1 WAR10 ELV Dafan SND1 WHM07 HUM Darie SND1 THF08 MIT Enoyaa SND1 WAR10 GAL Gotont SND1 BLM08 TAR Fegogo SND1 WHM08 TAR Yoteite Group 50 詳細(Detail) SND1 RDM14 HUM Alynn SND1 WHM14 TAR Deijii SND1 WAR14 ELV Kung SND1 BLM14 TAR Lehehe SND1 THF14 MIT Oomala SND1 WAR14 ELV Paude Group 51 詳細(Detail) BAS1 THF09 MIT Fittelly BAS1 RDM09 TAR Korunic BAS1 BLM10 TAR Stong BAS1 WAR11 HUM Thinver Kinmcosdy Mischievou Group 52 (削除) Group 53 詳細(Detail) Badgirls Mordys Songbing Tanshan Tonghua Xiaohei Group 54 詳細(Detail) (PL by Catsister) BAS1 ELV Frogmans(Alternative Character:Jorke/Basss) BAS1 TAR Huhhuh BAS1 TAR Jook BAS1 BLM6 TAR Basss BAS1 WAR7 HUM Bastt BAS1 BLM6 TAR Glacier SND1 THF25 MIT Eoundless Group 55 詳細(Detail) SND1 WHM04 TAR Akoi (LS WigSplitter) SND1 THF04 ELV Catlum SND1 WAR05 GAL Chroms SND1 BLM04 TAR Hoodlum SND1 WAR04 HUM Pandap SND1 BLM05 TAR Plmcypyx Group 56 詳細(Detail) SND1 BLM12 TAR Cheser SND1 WAR14 ELV Zoeobw SND1 THF13 MIT Leoparder SND1 WHM13 TAR Demonea SND1 RDM13 HUM Caesaris SND1 WAR14 ELV Freestyles Group 57 詳細(Detail) SND1 WAR09 HUM Meneedexp SND1 WHM09 TAR Rainaa SND1 RDM09 HUM Sevena SND1 THF09 MIT Wact Group 58 詳細(Detail) Blaakk SND1 WHM08 TAR Humama SND1 WAR12 HUM Longshao SND1 THF10 MIT Maonilili SND1 WAR11 ELV Ninkyl Olalamei Group 59 詳細(Detail) WIN1 WAR05 MIT Linta WIN1 BLM07 TAR Qutera Kyuzodono Mononey Ricecake Wiget Group 60 詳細(Detail) BAS1 WHM03 TAR Baiwhrryss BAS1 THF03 MIT Daouqthf BAS1 BLM03 TAR Hewherefind BAS1 RDM03 HUM Honxayssc BAS1 WAR03 ELV Rthvynftk BAS1 WAR03 ELV Rtsvynuftj Group 61 詳細(Detail) BAS1 BLM01 TAR Baipressisit BAS1 RDM01 HUM Hornredmage BAS1 WAR01 ELV Rtbgeneratefor Rtyopchoose Heigeeratradom Daogamemast Group 62 詳細(Detail) BAS1 THF11 MIT Chrizart Hontiost BAS1 WAR12 ELV Keelonly BAS1 RDM11 TAR Yukisally Patyoon Perfecter Group 63 詳細(Detail) BAS1 WAR05 ELV Insane BAS1 WAR05 ELV Goodtank BAS1 WHM05 TAR Xukiss BAS1 BLM05 TAR Summit BAS1 THF05 ELV Shaleless BAS1 RDM05 TAR Jackrain Group 64 詳細(Detail) SND1 BLM11 TAR Blockk SND1 WAR11 ELV Exits SND1 WHM11 HUM Kely SND1 THF12 MIT Sigee SND1 WHM12 TAR Minminnn Pleasee Group 65 詳細(Detail) (PL by Mindgrasp) SND1 WHM05 TAR Angleruba SND1 BLM05 TAR Casery SND1 WHM05 TAR Kingsbary SND1 WAR05 ELV Malimenlu SND1 THF05 MIT Misdom SND1 WAR05 HUM Nisery Group 66 詳細(Detail) SND1 WAR11 ELV Breezewarrior SND1 BLM11 TAR Darkemissary SND1 WAR11 ELV Firewarrior SND1 RDM09 TAR Soulemissary SND1 THF12 TAR Stealheart Nurseinwhite Group 67 詳細(Detail) SND1 BLM05 TAR Ayeganye SND1 WAR09 ELV Podant SND1 WAR10 ELV Vedrocent SND1 RDM05 TAR Kyaakuku SND1 THF06 MIT Tuhaa SND1 WHM05 TAR Tuyuluyu Group 68 詳細(Detail) SND1 BLM08 TAR Maxine SND1 BLM09 TAR Briange SND1 WAR08 ELV Brant SND1 RDM09 TAR Tarulife SND1 BLM07 TAR Anthenau SND1 BLM09 TAR Amandalu Group 69 詳細(Detail) SND1 WHM08 TAR Athenss SND1 WAR08 ELV Tinmyt SND1 WAR08 ELV Akamh SND1 THF09 MIT Mitiny SND1 THF09 MIT Tray SND1 BLM09 TAR Ytur Group 70 詳細(Detail) (PL SND5 BLM59/WHM29 TAR Mive) SND1 RDM14 HUM Jacob SND1 WAR15 HUM Jerome SND1 WHM14 TAR Kowayi SND1 BLM12 TAR Monoro SND1 THF14 MIT Botteye SND1 WAR15 ELV Gehry Group 71 詳細(Detail) BAS1 WHM12 TAR Banatutu BAS1 THF13 MIT Chihastaagger BAS1 WAR12 MIT Tentahha BAS1 RDM13 HUM Demiran BAS1 WAR13 ELV Docien BAS1 MNK13 ELV Fadrubion BAS1 THF13 MIT Hahpypy BAS1 BLM12 TAR Hiputniput BAS1 WAR13 GAL Holygorilla BAS1 MNK13 GAL Platedbonito BAS1 WHM13 TAR Yopolulu Minneline Group 72 詳細(Detail) SND1 WAR08 MIT Bees SND1 THF09 MIT Eady SND1 WHM08 MIT Nimi SND1 WAR08 MIT Trac SND1 WHM08 MIT Giny SND1 WHM07 MIT Mays Group 73 詳細(Detail) BAS1 BLM14 TAR Chenroc BAS1 WAR14 ELV Chossses BAS1 THF14 HUM Nakoo BAs1 RDM15 TAR Chumimii BAS1 WHM15 TAR Thercoat BAS1 WAR14 ELV Suzakua Group 74 詳細(Detail) SND1 WHM06 TAR Charisant Desksky Generated Jinsheng SND1 BLM06 TAR Mainicon Tanki Group 75 詳細(Detail) BAS1 WAR11 HUM Belinie BAS1 WAR12 MIT Byakitty BAS1 RDM11 TAR Mohabeha BAS1 WAR11 GAL Redekk BAS1 WHM12 TAR Riyosisi BAS1 WAR11 MIT Tsava Group 76 詳細(Detail) Keeley Pilaeee WIN1 RDM20 HUM Dall WIN1 BLM18 TAR Dasusu WIN1 THF19 MIT Eynes WIN1 WAR18 MIT Guh Group 77 詳細(Detail) SND1 WAR23 ELV Akubisama SND1 THF24 MIT Kristoperp SND1 WHM23 TAR Kuer Nokkochan Panazawa Peordes Group 78 詳細(Detail) SND1 WAR26/MNK05 GAL Cocboy SND1 WHM28/BLM07 TAR Cocgirl SND1 BLM26/WHM05 TAR Godisboy SND1 THF29/WAR05 MIT Warai Group 79 詳細(Detail) SND1 THF13 MIT Forlove SND1 THF13 MIT Gnndbya SND1 WHM11 TAR Goodchild SND1 WAR13 ELV Kjghjthtogr SND1 RDM13 HUM Rfufofeilu SND1 WAR13 ELV Dfyhjdklh Group 80 SND1 NIN35/WAR17 ELV Jaza SND1 WHM37/BLM18 TAR Bstkey SND1 THF37/NIN18 MIT Kahalera Solo or Small group MIT Avgirl ELV Bluelion SND2 THF59/NIN29 MIT Zxchiop Dedracent Pyoih Vebreaubent BAS2 NIN08/THF04 ELV Vublebaut BAS2 NIN08/WAR04 MIT Wwehe Japanese Group Selbinagodone Selbinagodtwo Selbinagodthree Selbinagodfour Selbinagodfive Selbinagodsix Selbinagodseven Former Group 1 詳細(Detail) TAR Angle HUM Bettylily ELV Calmikeboss TAR Everblack ELV Kriseman MIT Lamatilecat Former Group 2 詳細(Detail) Tuky Wantfly Jkf Idhdgd BAS6 NIN75/WAR37 ELV Gssyt Cows Former Group 3 詳細(Detail) Arcgood (LS SPongDaggers) Daly Junny WIN1 NIN75/WAR37 GAL Large WIN1 THF75/WAR12 MIT Luckyangel Maty Former Group 4 詳細(Detail) Icebird Kiriny TAR Taruanko MIT Tkfc SND5 BST60/WHM30 HUM Skyfenrir (for Search) SND6 PLD68/WAR34 GAL Keren Former Group 5 Houston Jazze Rimies Sasukeii Harunosakura Grammy Former Group 6 Beenyuyu Ngueh Nother Standby Former Group 7 Bimaoshua Hongmo Raisev Joooo Mengder Luchking Former Group 8 Anytal BAS10 RDM75/THF34 ELV Furyrush Whitra Yuzzi Vighht Krebs Altaar BAS10 DRK74/THF37 ELV Greenrose Luxuriant Shahalia Bihtsee Biluvy Biyot Firido Higgyu Motuman Sekuj Tukatu Asialamie Elv Bluelion Elv Amwamp Amwbi Amwgo Amoli Guro Leol Peterli Altaar Breezer Fireleopard Flypanda Flyzero Smalldrakan 合成 (Crafting) Zzxx Basdfg Skylong Vyyuke Yenge Ywiss Qubin Queking Quesm (LS KingdomOfHeroes) (Same group with Krebs) Quedage Quebbb Queoki Queoo Queyy Quehh Quexx Queccc Queddd Aquet Bquet Foque Bque Jque Shotque Tempque 採掘 (Minner) (Dead?)Newjack (Dead?)Newelbort Nathumin Wooky Flanna Dybyeth 釣り(Fishing) ELV Qusea ELV Quese MIT Quww Quwg Qugg Andycool Catsister Kinafees Littlepig Liudehua Mrcool MIT Pomaya MIT Pokoren MIT Divnoe MIT Sycdxx MIT Vbhnpof MIT Florana MIT Novie MIT Maspoko MIT Moonmage MIT Babcd MIT Xrcddsy MIT Sviod MIT Xgetx MIT Zyoo MIT Goher MIT Raisu MIT Mxqwiq MIT Jaileroflove MIT Kukbum 競売(Auction House) Qeoos Queoso Queasg Quetofiv Quejjj Quejmj Quejtj Quelll Quezz チョコボ掘り(Chocobo Digging) TAR Dussope (Rolanberry) TAR Equine TAR Rabufa (West Altepa Desert) Tualual (East Altepa Desert) Investigating/Cleaning up Asialamie, Asurathief, Avgirl, Alinabird, Abigale, Ajbi, Arcgood, Amoli, Arise, Angle, Altaar, Anytal Byotagyunuyi, Bettylily, Bluelion, Breezer, Beenyuyu, Bihtsee, Biluvy, Biyot, Bimaoshua, Basdfg Cows, Creaativ, Calmitkeboss Dayli, Daicajins, Dybyeth Elveengirl, Everbluck, Eowils Flytosky, Flyzero, Flypanda, Fireleopard, Firido, Furyush Goodmen, Gssyt, Grammy, Greenrose, Goodbey Harunosakura, Houston, Higgyu Iawxrwxf, Icebird, Idhdgd Jhkf, Jefrige Kiriny, Keren Leol, Leave, Loyayaya, Lamatilececat, Luxuriant, Luckyangel, Luky, Lanbant, Luxuriant Motuman, Mmqwer, Mmasdf, Mmzxcv, Mmnbv, MMlkjh Ngueh Pcl, Peterli Rimes, Raisev Serapher, Smalldrakan, Sali, Sasukeii, Sodepopo, Sakuj, Skylong, Sdvb Tuky, Taruanko, Tukatu Vighht Wantfly, Winddie, Whitra Yuio, Yjpejgg, Yuzzi Zzxx レンタル掲示板
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TOXICOLOGICAL PROFILE FOR WHITE PHOSPHORUS -index ▼2 HEALTH EFFECTS ▼▼2.2 DISCUSSION OF HEALTH EFFECTS BY ROUTE OF EXPOSURE 2.2.2 Oral Exposure -2 2.2.2 Oral Exposure(2.2.2.0 preface) 2.2.2.1 Death 2.2.2 Oral Exposure -22.2.2.2 Systemic Effects(preface) Respiratory Effects. Cardiovascular Effects. Gastrointestinal Effects. Hematological Effects. Musculoskeletal Effects. Hepatic Effects. Renal Effects. Dermal Effects. Other Systemic Effects. 2.2.2 Oral Exposure -32.2.2.3 Immunological and Lymphoreticular Effects 2.2.2.4 Neurological Effects 2.2.2.5 Reproductive Effects 2.2.2.6 Developmental Effects 2.2.2.7 Genotoxic Effects 2.2.2.8 Cancer 2.2.2.2 Systemic Effects (preface) Systemic effects of white phosphorus in humans and animals after oral exposure are discussed below. The highest NOAEL value and all reliable LOAEL values from each reliable study for systemic effects in each species and duration category are recorded in Table 2-2 and plotted in Figure 2-2. No studies were located regarding ocular effects in humans or animals after oral exposure to white phosphorus. Respiratory Effects. Studies on respiratory effects following acute oral exposure of humans to white phosphorus were limited to case reports of intentional or accidental consumption of materials containing white phosphorus. Although intake of phosphorus was often reported, dose could be estimated for only one study (Hann and Veale 1910), because vomiting and/or gastric lavage nearly always occurred soon after poisoning, expelling much of the ingested phosphorus from the body. Tachypnea (increased respiratory rate; 48 breaths/minute) was observed in a woman consuming rat poison containing 4% white phosphorus (Hann and Veale 1910); the woman apparently did not vomit until the second day, and the vomitus was clear. The estimated dose was 2 mg/kg. Four days after ingesting the rat poison, the woman died, apparently from liver failure. Autopsy showed that the pleural cavity was filled with a dark fluid, but no histological abnormalities were observed in the lungs (Hann and Veale 1910). In the following studies, no doses could be estimated for respiratory effects because of vomiting and/or gastric lavage. In a case report involving ingestion of rat poison containing white phosphorus, the patient arrived at a hospital in a coma and displayed Cheyne-Stokes respirations and rales (Wechsler and Wechsler 1951). The Cheyne-Stokes respirations increased to an extreme degree, and the patient died. Autopsy revealed pulmonary congestion and edema throughout the stroma. Increased respiratory rate (56 breaths/minute) and rales also were observed in an infant ingesting rat poison containing white phosphorus (Rao and Brown 1974). The child died, and autopsy revealed evidence of pulmonary edema. Rales were observed in a child ingesting a fatal dose of white phosphorus in fireworks; autopsy indicated that the lungs were normal except for some fibrous adhesions (Dwyer and Helwig 1925). Hemorrhagic bronchopneumonia was observed following autopsy of a man ingesting a fatal dose of rat poison containing white phosphorus (Winek et al. 1973). Autopsy of a child who died following ingestion of a firecracker revealed fatty deposition in parenchyma, bronchial epithelium, and tracheal epithelium and cartilage (Humphreys and Halpert 1931). Death from cardiopulmonary failure was reported for a 63-year-old woman (Winek et al. 1973), a 2-year-old boy (Simon and Pickering 1976), and a 3-year-old girl (Simon and Pickering 1976) following ingestion of white phosphorus in rat poison; a respiratory rate of 44 breaths/minute was initially observed in the girl (Simon and Pickering 1976). Increased respiratory rate was observed prior to death in two case reports involving ingestion of rat poison (Talley et al. 1972; Winek et al. 1973). Shallow respirations and cyanosis were observed prior to death in an adult female following ingestion of rat/roach poison containing white phosphorus (Rubitsky and Myerson 1949). Rales were observed 1 day after intentional ingestion of rat poison by a 30-year-old man; 2 days later the patient went into shock but survived the poisoning and eventually recovered (Pietras et al. 1968). No treatment-related respiratory effects were reported in children treated with white phosphorus for intermediate durations. No treatment-related microscopic changes were observed in the lungs of rats exposed to 0.2 mg/kg/day white phosphorus in the diet for a chronic duration (Fleming et al. 1942) or 0.075 mg/kg/day phosphorus by gavage for an intermediate duration (LRDC 1985). Heavy breathing and apnea were reported following ingestion of a fatal quantity of white phosphorus by a cat (Frye and Cucuell969). Necropsy revealed hyperemia, hemorrhage and edema in the lungs. Cardiovascular Effects. Alterations in electrocardiograms, such as altered or inverted T waves and changes in the QRS complex, and other cardiac changes, such as tachycardia, arrhythmias, atrial fibrillation, and decreased ventricular contractility, have been observed in individuals accidentally or intentionally ingesting a single dose of white phosphorus (Dathe and Nathan 1946; Diaz-Rivera et al. 1950, 1961; Dwyer and Helwig 1925; Ehrentheil 1957; Matsumoto et al. 1972; McCarron et al. 1981; Newburger et al. 1948; Pietras et al. 1968; Rao and Brown 1974; Simon and Pickering 1976; Talley et al. 1972). Damage to the myocardium was verified by a number of cases in which histological examination of the heart was performed. Prominent cross striations in the myocardium (Dwyer and Helwig 1925), fatty infiltration of muscle (Diaz-Rivera et al. 1961; Humphreys and Halpert 1931; Wertham 1932), necrosis of myocardium (Wechsler and Wechsler 1951), markedly dilated cardiac chamber (Rao and Brown 1974), and interstitial edema of the myocardium and vacuolation of cells (Talley et al. 1972) have been observed. Because of vomiting and gastric lavage, doses cannot be calculated from the human studies. No cardiac effects were reported in longer term human studies. In addition to the effects on the heart, a number of vascular effects have been observed in humans acutely exposed to white phosphorus. A markedly decreased or undetectable blood pressure (Caley and Kellock 1955; Dathe and Nathan 1946; McCarron et al. 1981; Rubitsky and Myerson 1949; Simon and Pickering 1976; Wechsler and Wechsler 1951), vascular collapse (Diaz-Rivera et al. 1950, 1961), undetectable or decreased pulse (Dwyer and Helwig 1925; Rubitsky and Myerson 1949), and increased pulse (Dathe and Nathan 1946; Hann and Veale 1910; McCarron et al. 1981; Wechsler and Wechsler 1951) have been observed. In addition, individuals have died following cardiopulmonary arrest (Simon and Pickering 1976; Winek et al. 1973), which may be due to effects on the heart or vascular system. A dose of 2 mg/kg/day for vascular effects was identified from the Hann and Veale (1910) report of a woman ingesting a single dose of white phosphorus. Dose levels cannot be estimated for the other case reports. Hemorrhaging in internal organs, as well as the appearance of petechial hemorrhages on the skin, have been reported in a number of acute human exposure cases (Hann and Veale 1910; Humphreys and Halpert 1931; Winek et al. 1973). It is not known whether these effects are due to impairment of the integrity of the blood vessels or due to damage of the affected organ (e.g., liver, stomach) itself. In rats administered 0.075 mg/kg/day white phosphorus for an intermediate duration, no histological alterations were observed in the heart (Bio/dynamics 1991; IRDC 1985). In rats exposed for an intermediate duration to an unknown concentration of airborne white phosphorus from the furnace room of a phosphorus factory, an increase in permeability of capillary walls, lesions in the walls of blood vessels and evidence of impaired microcirculation were observed in the mouth (Ruzuddinov and Rys-Uly 1986). These effects probably resulted from the local action of white phosphorus on the oral cavity. Gastrointestinal Effects. Most of the human case reports listed vomiting as an early effect following ingestion of a single high dose of white phosphorus (Caley and Kellock 1955; Dathe and Nathan 1946; Diaz-Rivera et al. 1950; Dwyer and Helwig 1925; Ehrentheil 1957; Fletcher and Galambos 1963; Greenberger et al. 1964; Hann and Veale 1910; Humphreys and Halpert 193 1; Matsumoto et al. 1972; McCarron et al. 1981; McIntosh 1927; Newburger et al. 1948; Pietras et al. 1968; Rubitsky and Myerson 1949; Simon and Pickering 1976; Wechsler and Wechsler 1951; Winek et al. 1973). The doses that induced vomiting ranged from 2 to 23 mg/kg (Caley and Kellock 1955; Ehrentheil 1957; Fletcher and Galambos 1963; Harm and Veale 1910; Matsumoto et al. 1972; McCarron et al. 1981; Newburger et al. 1948; Rubitsky and Myerson 1949). Vomiting generally started within hours after ingesting the white phosphorus, and sometimes continued for many days. Other gastrointestinal effects included abdominal cramps or pain (often severe) (Dwyer and Helwig 1925; Ehrentheil1957; Fletcher and Galambos 1963; Greenberger et al. 1964; Humphreys and Halpert 1931; McCarron et al. 1981; Newburger et al. 1948; Pietras et al. 1968), vomiting blood and/or pieces of the gastric mucosa (Dathe and Nathan 1946; Diaz-Rivera et al. 1950; Rubitsky and Myerson 1949), necrosis and erosion of mucosa in the esophagus, stomach, duodenum, and jejunum (Wechsler and Wechsler 1951), and gastrointestinal hemorrhage (Dwyer and Helwig 1925; Hann and Veale 1910; Humphreys and Halpert 1931; Wertham 1932, Winek et al. 1973). These effects, with the exception of necrosis, were probably due to the irritating effects of white phosphorus on the mucosa of the gastrointestinal tract. Vomitus often contained white phosphorus, indicating that vomiting generally occurred before all white phosphorus and/or its oxidation products had been absorbed. No gastrointestinal effects were reported in children receiving treatment with 0.026-0.158 mg/kg/day white phosphorus for as much as 26 months (Phemister 1918; Compere 1930a). An infant became seriously ill during treatment with 0.083 mg/kg/day white phosphorus (6-month time-weighted average dose), but recovered entirely following discontinuation of the treatment (Sontag 1938). No vomiting or diarrhea was observed during the treatment period. Gastrointestinal effects were not reported in studies examining longer term occupational exposure to white phosphorus (Heimann 1946; Hughes et al. 1962; Kennon and Hallam 1944; Ward 1928). Erosion and hemorrhages in tissue in the esophagus and stomach were observed following ingestion of a fatal unknown quantity of white phosphorus by a cat (Frye and Cucuel 1969). Vomiting was observed in 6 of 21 dogs treated by gavage with an unknown quantity of white phosphorus from firecrackers (Dwyer and Helwig 1925). No gross or microscopic alterations were observed in the gastrointestinal tract of rats treated by gavage with 0.075 mg/kg/day for 204 days (IRDC 1985). Hematological Effects. Hematological effects have been reported in a number of case histories of individuals accidentally or intentionally ingesting a single dose of white phosphorus contained in rat (and cockroach) poisons or fireworks. Because most of the individuals vomited or received gastric lavage shortly after ingestion, the amount of white phosphorus available for absorption is not known. Increases in erythrocyte levels (Diaz-Rivera et al. 1950) and hemoglobin levels (Diaz-Rivera et al. 1950; McIntosh 1927); decreases in erythrocyte levels (Dwyer and Helwig 1925) and hemoglobin and/or hematocrit levels (Simon and Pickering 1973); and anemia (Caley and Kellock 1955) have been observed in some of these individuals. A number of individuals had no change in erythrocyte parameters (Ehrentheil 1957; Fletcher and Galambos 1963; Newburger et al. 1948; Simon and Pickering 1976). The decreases in erythrocyte parameters may be a reflection of the hemorrhages observed in specific tissues (e.g., gastrointestinal tract, liver, skin) (Dathe and Nathan 1946; Hann and Veale 1910; Humphreys and Halpert 1931; Wechsler and Wechsler 1951; Winek et al. 1973). In addition to these changes in erythrocyte parameters, changes in total or differential leukocyte levels were reported in a number of individuals acutely exposed to white phosphorus. Decreases in total leukocyte levels (Diaz-Rivera et al. 1950; Ehrentbeil 1957; Fletcher and Galambos 1963; McCarron et al. 1981; Newburger et al. 1948; Pietras et al. 1968) and decreases or increases in the percentage of polymorphonuclear leukocytes (neutrophils) have been reported (Ehrentheil 1957; McCarron et al. 1981; Pietras et al. 1968). No changes in leukocyte parameters were observed in a number of individuals (Fletcher and Galambos 1963; Newburger et al. 1948; Simon and Pickering 1976). Abnormally low protbrombin times or levels (hypo-prothrombinemia) and a moderate decrease in platelets were observed in a number of individuals ingesting single doses of white phosphorus (Caley and Kellock 1955; Dathe and Nathan 1946; Ehrentheil 1957; Fletcher and Galambos 1963; McCarron et al 1981). Most of the patients developed hypoprothrombinemia within 4-8 days (McCarron et al. 1981). This is probably secondary to the liver damage rather than a direct effect on platelets. No changes in hematological parameters were observed in a child ingesting phosphorized cod liver oil (0.083 mg/kg/day phosphorus) for 184 days (Sontag 1938). Anemia and leukopenia were observed in individuals occupationally exposed to white phosphorus chronically (Ward 1928). It is likely that workers were exposed by the inhalation, oral, and dermal routes. Because there is very little consistency regarding the length of time that elapsed between ingestion and measurement of hematological parameters and the doses cannot be calculated, it is difficult to compare the results of different studies. There is insufficient information to determine whether white phosphorus has a direct effect on erythrocytes and/or leukocytes. The effects observed may be secondary effects. The decrease in erythrocyte, hemoglobin, hematocrit and leukocyte levels may be secondary to hemorrhaging or hematoemesis (Diaz-Rivera et al. 1950; Rubitsky and Myerson 1949) and the increase in erythrocytes and hemoglobin may be a compensatory mechanism due to tissue anoxia. However, since red blood cell synthesis takes 3-5 days, the observed effects may be direct if they are occurring within l-2 days. A slight decrease in hemoglobin levels and increase in eosinophil levels were observed in a 30-year-old man who performed magic shows that involved placing white phosphorus pellets in the mucobuccal folds of his mouth for 15 years. He had no other personal habits that might adversely affect his health except for occasional bidi smoking for about 8 years (Jakhi et al. 1983). Information on hematological effects in animals is limited to one study in which a marked increase in total leukocyte levels and the percentage of monocytes were observed in a guinea pig acutely exposed to 0.9-2.4 mg/kg/day of white phosphorus in a complex dosing regimen (Lawrence and Huffman 1929). The study authors did not specify at which doses the effects occurred. Musculoskeletal Effects. Following ingestion of a fatal dose of rat poison containing white phosphorus by a woman, autopsy revealed fatty infiltration of essentially all tissues, including the musculature (Wertham 1932). Similar effects were reported following the death of a male child who accidentally ingested a firecracker containing white phosphorus; autopsy revealed fatty deposition in many tissues, included the diaphragmic muscle (Humphreys and Halpert 1931). Humans occupationally exposed to white phosphorus probably ingested some airborne white phosphorus. In a study of 71 humans occupationally exposed to white phosphorus, oral exposure to white phosphorus via hand-to-mouth activity was likely because the workers constantly handled a paste containing 4-6% white phosphorus and washroom facilities were inadequate (Ward 1928). In workers exposed to white phosphorus for intermediate durations, 2 of 44 developed phossy jaw, described as slight necrosis in the lower jaw. In workers exposed to white phosphorus for chronic durations, 12 of 27 developed phossy jaw, with necrosis ranging from slight to severe; 2 of the 12 workers developing phossy jaw died from complications related to the necrosis. The progression of the disease was similar in the cases described, usually beginning with the extraction of one or more teeth, poor healing of the socket, followed by necrosis of tissue in the jaw with severe pain and infection. Treatment consisted of repeated removal of destroyed bone tissue and teeth, draining of abscesses, and reconstructive surgery. In severe cases, extensive removal of necrotic bone tissue led to permanent disfigurement. However, exposure levels of white phosphorus were not reported (Ward 1928). Case reports of development of phossy jaw following intermediate or chronic occupational exposure to unreported levels of white phosphorus and phosphorus compounds describe a similar progression of symptoms, with similar results; even in cases of early diagnosis and prompt, intensive treatment of phossy jaw, recovery often took several years (Heimann 1946; Hughes et al. 1962; Kennon and Hallam 1944). It is likely that the effect of white phosphorus in the oral cavity is local, resulting from contact of “inhaled”white phosphorus particles with tissue in the mouth. White phosphorus may affect the oral mucosa. Dull, red spots in the oral mucosa, an early sign of phossy jaw, have been reported to precede its development in occupationally exposed workers (Kennon and Hallam 1944). The oral mucosa of workers exposed to white phosphorus has been described as having a dull, red, unhealthy appearance (Hughes et al. 1962). Exposed bones may be especially susceptible to the irritating affects of white phosphorus. It is not known whether white phosphorus ingested and absorbed into the systemic circulation contributed to the development of phossy jaw. Not all workers exposed to white phosphorus for longer-term durations developed phossy jaw. In a study of 71 workers exposed to airborne white phosphorus for intermediate or chronic durations, 4.5% and 44%, respectively, developed phossy jaw (Ward 1928). Forty-eight male workers with exposure to white phosphorus ranging from 1 to 17 years were found to be normal and healthy with regards to many parameters, including serum levels of calcium and phosphorus, and bone density; none of the men developed phossy jaw (Hughes et al. 1962). Tooth loss often precedes and accompanies the progression of development of phossy jaw (Heimann 1946; Hughes et al. 1962; Kennon and Hallam 1944; Ward 1928). Tooth loss during the later stages of phossy jaw clearly results from destruction of the-bone structure supporting the teeth (Heimann 1946; Hughes et al. 1962; Kennon and Hallam 1944; Legge 1920; Ward 1928). It is not known if tooth loss prior to diagnosis of phossy jaw or early in the development of the condition is related to the white phosphorus exposure. Poor dental hygiene alone can result in tooth loss, and in several case reports some of the workers were described as having poor dental hygiene (Kennon and Hallam 1944). Tooth loss followed by poor healing of the socket often precedes development of the necrosis (Heimann 1946; Hughes et al. 1962; Kennon and Hallam 1944; Ward 1928), suggesting that poor dental hygiene and exposure to white phosphorus may both be contributing factors to the development of phossy jaw. In a case report, five men developed “precursor signs” (delayed healing of extracted tooth sites and residual sepsis) of phossy jaw developed following tooth extraction and occupational exposure to white phosphorus (Hughes et al. 1962). However, the condition did not develop into “classical” phossy jaw. A man was repeatedly exposed to white phosphorus pellets, placed in the right mucobuccal cavity for magic shows, for . 15 years (Jakhi et al. 1983). After . 14.5 years of this type of exposure to white phosphorus, right maxillary molars became loose, and were subsequently lost. This was followed by a lack of healing and development of fistulae in the sockets of the right maxillary molars. White phosphorus necrosis of the jaw developed, with massive necrosis of the maxilla and floor of the antrum on the right side of the mouth; perforations were present through which the maxillary sinus and nasal cavity were visible. No effects were observed on the left side of the maxilla or on the mandible. Radiographs revealed no evidence of pathology in the chest and long bones. The damage to the jaw was probably caused by direct local contact of phosphorus with the soft tissue and bone in the oral cavity. No microscopic or histological abnormalities were observed in the bone of rats treated by gavage with 0.075 mg/kg/day for 204 days (IRDC 1985). Rats exposed for a chronic duration to 0.2 mg/kg/day white phosphorus in the diet had epiphyseal line thickening and greater extension of trabeculae into the diaphysis of unspecified bones, compared to a control group (Fleming et al. 1942). This study is limited by the failure to specify incidences of effects at interval during dosing and by the failure to state the dosing duration explicitly. Bone effects were observed in children (Compere 1930a; Phemister 1918; Sontag 1938) and young animals (Adams 1938a, 1938b; Adams and Sarnat 1940; Whalen et al. 1973) following acute and intermediate oral exposure to white phosphorus. Because white phosphorus-related effects were observed in growing bones, these effects were considered developmental effects, and are discussed in. Section 2.2.2.6. Hepatic Effects. Hepatic effects have been observed in most individuals accidentally or intentionally ingesting a single dose of white phosphorus. These effects include jaundice (Caley and Kellock 1955; Diaz-Rivera et al. 1950, 1961; Ehrentheil 1957; Greenberger et al. 1964; Humphreys and Halpert 1931; McCarron et al. 1981), hepatomegaly (Diaz-Rivera et al. 1950; Fletcher and Galambos 1963; Humphreys and Halpert 1931; Rao and Brown 1974; Simon and Pickering 1976; Wechsler and Wechsler 1951), increased levels of serum bilirubin (Caley and Kellock 1955; Fletcher and Galambos 1963; McCarron et al. 1981; Pietras et al. 1968), impaired liver function test results (Fletcher and Galambos 1963; Newburger et al. 1948; Pietras et al. 1968; Rubitsky and Myerson 1949), and increases in AST, ALT, and/or lactate dehydrogenase (Ehrentheil 1957; Matsumoto et al. 1972; McCarron et al. 1981; Pietras et al. 1968). In addition to these effects, autopsies or liver biopsies have revealed a number of histological alterations in these individuals. Necrosis (Fletcher and Galambos 1963; Rao and Brown 1974; Wechsler and Wechsler 1951), degeneration (Dwyer and Helwig 1925; Greenberger et al. 1964; Wechsler and Wechsler 1951), fibrosis (Greenberger et al. 1964), hemorrhages (Wechsler and Wechsler 1951), and fatty infiltration (Dwyer and Helwig 1925; Hann and Veale 1910; Humphreys and Halpert 1931; Wertham 1932) have been observed in the liver. In addition to these effects, altered prothrombin time or level has been observed in a number of individuals ingesting a single dose of white phosphorus (Caley and Kellock 1955; Dathe and Nathan 1946; Ehrentheill957; Fletcher and Galambos 1963; McCarron et al. 1981). Prothrombin and other plasma proteins that are required for the efficient progression and regulation of blood coagulation are primarily synthesized in the liver. A deficiency of these proteins is often observed in individuals with severe liver disease. A prolongation of prothrombin time is in part due to this impaired synthesis. Liver function tests were normal in workers chronically exposed to unreported levels of airborne phosphorus (Hughes et al. 1962). Similar hepatic alterations have been observed in animals acutely exposed to white phosphorus. Increases in AST and ALT levels (Paradisi et al. 1984), impaired liver function tests (Ghoshal et al. 1969; Hurwitz 1972; Sigal et al. 1954) increased liver weight (Ghoshal et al. 1969; Seakins and Robinson 1964), increased hepatic triglyceride levels (Ghoshal et al. 1969; Pani et al. 1972; Paradisi et al. 1984; Seakins and Robinson 1964), decreased protein synthesis (Barker et al. 1963; Seakins and Robinson 1964), disaggregation of polyribosomes (Pani et al. 1972), fatty degeneration (Ghoshal et al. 1969) and necrosis (Ghoshal et al. 1969) have been observed. No NOAEL values for hepatic effects following acute animal exposure were identified. In rats, the LOAEL value for liver effects was 6 mg/kg (Barker et al. 1963); in mice it was 5 mgkglday (Hurwitz 1972); and in dogs it was 0.2 mg/kg/day (Sigal et al. 1954). The liver effects occurred shortly after dosing; 3 hours after dosing, a significant decrease in protein synthesis was observed in the liver (Barker et al. 1963), minimal hepatocytic fatty changes were observed after 4 hours (Ghoshal et al. 1969), and severe hepatocytic fatty changes were observed after 12 hours (Ghoshal et al. 1969). The following hepatic effects have been observed in animals orally exposed for an intermediate duration fatty infiltration in guinea pigs exposed to 0.75 mg/kg/day (Ashbum et al. 1948), presence of eosinophilic granules at 0.25 mg/kg/day and cirrhosis at 0.66 mg/kg/day in rabbits and guinea pigs (Mallory 1933), and fibrosis and cirrhosis in pigs exposed to 0.6 mg/kg for 5 days/week (Peterson et al. 1991). In the Peterson et al. (1991) study, no liver effects were observed after 4 weeks of exposure; after 8 weeks, there were early signs of fibrosis, and after 12 weeks, extensive fibrosis was observed. Exposure to 0.075 mg/kg/day for an intermediate duration resulted in slight-to-moderate liver necrosis in dying pregnant rats (Bio/dynamics 1991), but no hepatic effects in the surviving pregnant rats or in male rats (Bio/dynamics 1991). In another reproduction study, liver effects were not observed in dying pregnant rats exposed to 0.075 mg/kg/day (IRDC 1985). Both studies used similar exposure protocols and similar vehicles; the difference in the occurrence of liver damage between the studies cannot be explained. Renal Effects. Evidence of severe renal effects have been observed in a number of individuals intentionally or accidentally ingesting a single dose of white phosphorus contained in rat (or roach) poison or fireworks. Proteinuria (Matsumoto et al. 1972; Pietras et al. 1968; Rao and Brown 1974), albuminuria (Dathe and Nathan 1946; Diaz-Rivera et al. 1950; Dwyer and Helwig 1925; Fletcher and Galambos 1963; McCarron et al. 1981; Rubitsky and Myerson 1949), acetonuria (Pietras et al. 1968), increased urobilinogen (Matsumoto et al. 1972), oliguria (Dathe and Nathan 1946; McCarron et al. 1981; Rao and Brown 1974), increased blood levels of urea and/or nitrogen (Diaz-Rivera et al. 1950, 1961; McCarron et al. 1981; Newburger et al. 1948; Pietras et al. 1968; Rao and Brown 1974; Rubitsky and Myerson 1949), and increased blood creatinine levels (Dathe and Nathan 1946; McCarron et al. 1981) have been observed in these individuals. Renal insufficiency may be due to a direct toxic effect of phosphorus on the kidneys or to acute renal tubular necrosis from fluid loss and shock. Patients in shock may have a peculiar pallor and cyanosis. These probably reflect extensive cellular damage with poor perfusion of the capillary beds, and are a prognostic sign of serious health effects (Melamon et al. 1981). Several case reports have reported no alterations in kidney function (Ehrentheil 1957; Fletcher and Galambos 1963; Greenberger et al. 1964; Simon and Pickering 1976). Histological alterations have also been observed in a number of humans ingesting a single dose of white phosphorus. Fatty changes in the tubules and loop of Henle (Dwyer and Helwig 1925; Humphreys and Halpert 1931; Wertham 1932) and engorged glomeruli and intratubular capillaries (Wechsler and Wechsler 1951) have been observed. Because most individuals vomited shortly after ingesting the white phosphorus or were lavaged, accurate doses cannot be calculated except for one study (Harm and Veale 1910). Histological alterations in the kidney were observed in an individual ingesting 2 mg/kg/day, but the lesion was not described. Creatinine levels were similar among unexposed workers and workers exposed to white phosphorus for chronic durations (Hughes et al. 1962). In animals, fatty infiltration in the nephron and subcapsular hemorrhages were observed in dogs acutely exposed to an unspecified amount of white phosphorus (Dwyer and Helwig 1925). No renal effects were observed in rats exposed to 0.075 mg/kg/day for an intermediate duration (Bio/dynamics 1991; IRDC 1985). No chronic exposure animal studies examining renal effects were located. Dermal Effects. Transient toxic dermatitis (described as a scalartiniform rash) developed 9 days after a man ingested a near-fatal dose of rat poison (Dathe and Nathan 1946). Edema of eyelids was reported in a 13-month-old child after ingestion of a fatal dose of white phosphorus (Rao and Brown 1974). Subcutaneous hemorrhages were visible in the left foot in a woman after consumption of 3.9 g of rat poison containing 4% phosphorus (Hann and Veale 1910). The woman died 4 days after the initial poisoning. At this time, an enormous subcutaneous hemorrhage was visible below the waist line. In this case report, the woman apparently did not expel (via vomiting) any of the ingested dose. Thus, it is likely that the ingested dose (2 mg/kg) was representative of the effective dose. Scattered blue-green petechiae were observed on the abdomen of a male child following accidental ingestion of a fatal dose of white phosphorus mixed with other ingredients from a firecracker (Humphreys and Halpert 1931). The dose level in this study could not be determined; the firecracker was a red composition of phosphorus mixed with other ingredients and was thought to contain about 10% phosphorus (Humphreys and Halpert 1931). No studies were located regarding dermal effects in animals after oral exposure to white phosphorus. Other Systemic Effects. A number of other systemic effects have been observed in humans ingesting a single dose of white phosphorus. The effects that are observed most consistently are hypoglycemia (Diaz-Rivera et al. 1950; McCarron et al. 1981; McIntosh 1927; Wechsler and Wechsler 1951), an increase in body temperature (mild pyrexia or fever) (Dathe and Nathan 1946; McIntosh 1927), and a decrease in plasma calcium, potassium, and/or sodium levels (Caley and Kellock 1955; McCarron et al. 1981; Rao and Brown 1974). It is unclear whether the fever seen is a symptom of phosphorus poisoning or a result of the treatment involved. In addition to these effects, metabolic acidosis (Rao and Brown 1974), hypothermia (Simon and Pickering 1976), damage to the spleen (Greenberger et al. 1964), ascites (Fletcher and Galambos 1963), fatty infiltration of the pancreas (Humphreys and Halpert 193 l), and necrosis of the adrenal medulla and cortex (Wechsler and Wechsler 1951) have been observed. In a child ingesting 0.083 mg/kg/day white phosphorus for an intermediate duration, decreased appetite, impaired body weight gain, and poor turgor (fullness or tension produced by the fluid content of blood vessels, capillaries, and cells) were observed (Sontag 1938). Serum glucose levels were decreased in workers occupationally exposed to white phosphorus for a chronic duration. It is likely that the workers were exposed by the inhalation, oral, and dermal routes (Ward 1928). In dogs acutely exposed to an unspecified amount of white phosphorus, hypoglycemia was observed (Williamson and Mann 1923). Rats received intermittent exposure to the atmosphere in the furnace room of a phosphorus factory for 14 months (Ruzuddinov and Rys-Uly 1986). Histology of rats killed monthly revealed progressive morphological degeneration of the tongue and oral mucosa of the cheek, gum, and hard palate. Epithelium and connective tissue from different parts of the oral cavity responded similarly to the treatment. Changes in the epithelial layer, observed after only 1 month of exposure, included increases in keratinization and numbers of cell layers, resulting in thickening and hyperkeratosis in the epithelium of the mucosa. Over time, the thickening and hyperkeratosis in the epithelium increased and histological changes were observed in the subepithelial connective tissue base. Eventually, the oral cavity contained areas of thickening of the mucosa from hyperkeratosis and increased epithelial cell layers interspersed with areas of decreased thickness of the epithelial layer due to atrophy, dystrophy, and cellular necrosis. At this time, adverse changes in the subepithelial connective tissue were considered pronounced. These effects occurred in most of the animals exposed to the atmosphere. It is likely that the observed effects of phosphorus on the oral cavity were local rather than systemic, resulting from direct contact of white phosphorus with tissues in the mouth. The study presented essentially no quantitative data, and the types and exposure levels of chemicals in the atmosphere (thought to contain elementary phosphorus and its inorganic compounds) were not reported (Ruzuddinov and Rys-Uly 1986). 2.2 DISCUSSION OF HEALTH EFFECTS BY ROUTE OF EXPOSURE
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Cynosure Razor [部分編集] blankimgプラグインエラー:ご指定のファイルがありません。アップロード済みのファイルを指定してください。 Category Unique Item Class Templar Type Sword Item Level 31 Require Level 24 Damage Type Spectral Direct 55-82 Speed Slow Shield Overload 204% Interrupt Strength 165 Critical Chance 0% Critical Damage 0% Range 2m Phase Attack Strength 175 Equip Cost 59 Will, 11 Accu Modification Battery(1) Relic(1) Inherent Attributes Shield Overload 110% Special Attributes Increases Damage by 26% Decreases power cost of swordmanship skills by 17% Shield Overload +94% Increases Spectral Damage by 22% ※上記に画像がないときはアイテム名.jpgでアップロードしてください。 [部分編集] Special Attributes 固定 Affix名 効果 Divine? Increases Damage by 26% Miserly? Decreases power cost of swordmanship skills by 17% Demolishing? Shield Overload +94% Radiant? Increases Spectral Damage by 22% [部分編集] ・追加画像 関連ページ Beast of Abbadon Phase Blade Swords Unique Templar Weapons