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Vehicle Hetzer Contents 1 Hetzer Veterancy 2 Tactics 3 History 4 Requires 4.1 Hetzer Tank 5 Called In Using 5.1 Hetzer 6 Vehicle Abilities 6.1 Vehicle Cover 6.2 Ambush Lock-Down 7 Vehicle Upgrades 7.1 APCR Rounds 7.2 Spotting Scope 8 Vehicle Weapons 8.1 75mm Hetzer 8.2 75mm Hetzer APCR 8.3 MG42 Hetzer Hetzer Health 450 Max Speed 6.2 Sight 35 Cost 300 Acceleration 1.3 Detection 0/0 Time 30 Deceleration 2.75 Hotkey Population 8 Rotation 27 Target Type armour_hetzer Upkeep 1.0752 Crush Human true Critical Type armour Crush Mode crush_medium Rear Damage Enabled true Hetzer Veterancy [Expand][Hide] Received Damage Received Accuracy Maximum Speed Maximum Health 0.9 0.9 1.15 1.15 Accuracy Reload Cooldown Penetration 1.15 0.9 0.9 1.15 24 Vet-Exp Received Damage Received Accuracy Maximum Speed Maximum Health 0.95 0.95 1.05 1.15 Accuracy Reload Cooldown Penetration 1.15 0.9 0.9 1.15 58 Vet-Exp Received Damage Received Accuracy Maximum Speed Maximum Health 0.95 0.95 1.05 1.15 Accuracy Reload Cooldown Penetration 1.15 0.9 0.9 1.15 102 Vet-Exp Tactics The Hetzer can camoflauge when stationary and so, is good for ambushing enemy tanks raids. History The Jagdpanzer 38 (known after World War II as the Hetzer ( baiter )), was a German tank destroyer of the Second World War based on a modified pre-war Czechoslovakian Panzer 38 chassis. The Jagdpanzer 38 fitted into the lighter category of German tank destroyers that began with the Panzerjäger I, continued with the Marder series, and ended with the Jagdpanzer 38(t). The Hetzer was fitted with a modified 75mm Pak 39 L/48, very similar to the late Panzer IV s 75mm kwk 40 L/48; it could destroy nearly all allied tanks in service at longe range, and its fully-enclosed armor made it a much safer vehicle to crew than the Marder II or Marder III series. The Jagdpanzer 38(t) is one of the most common late-war German tank destroyers. It was crucially available in numbers that made it significant, and was generally mechanically reliable. Also, its small size made it a hard target, and was easy to conceal, allowing it to lie in ambush for enemy tanks. The Jagdpanzer 38(t) s weaknesses were its very limited gun traverse, poor internal ergonomics and poor visibility. The gun traverse was so limited that the entire vehicle sometimes needed to be turned to track a fast-moving target. The gun was designed to be loaded from the right but was also placed on the far right of the vehicle, making operation difficult for the gunner and loader, leading to a lower rate of fire than would be ideal. The confines of the vehicle were also very cramped, with four men squeezed into the small machine. The commander sat far back in the vehicle, with a flat roof to his front and without a cupola. Thus, his visibility was limited when the vehicle was even slightly elevated in back as, for example, in a classic hull-down position. Some versions attempted to alleviate the space problem by removing the recoil-absorbing mechanism for the gun, though it was at the cost of forcing the vehicle itself to absorb the recoil. Requires Hetzer Tank [Expand][Hide] Cost 3 Time Hotkey Effects A Hetzer Tank Hunter is available for reinforcement. These maneuverable tanks have thick sloping armor, and their small size means they are easily camouflaged. ESee Upgrade Hetzer Tank for details. Called In Using Hetzer [Expand][Hide] Cost 600 Activation targeted Duration 0 Target tp_any Recharge 40 Hotkey Effects The diminutive Hetzer mounted the same powerful Pak39 as used on the Panzer IV in well-armored chassis that was also small enough to easily conceal. ESee Ability Hetzer for details. Vehicle Abilities Vehicle Cover [Expand][Hide] Cost Activation always_on Duration _ Target tp_any Recharge 0 Hotkey Effects $0 no key ESee Ability Vehicle Cover for details. Ambush Lock-Down [Expand][Hide] Cost Activation toggle Duration 0 Target tp_any Recharge 10 Hotkey C Effects Lock-down your Hetzer and enter a Camouflaged state. The Hetzer s first-strike out of Camouflage has powerful ambush bonuses. ESee Ability Ambush Lock-Down for details. Vehicle Upgrades APCR Rounds [Expand][Hide] Cost 3 Time Hotkey Effects AT guns and main tank guns are issued the more powerful APCR (Armor Piercing Composite Rigid) Panzergranate 40 ammunition that will improve armor penetration. ESee Upgrade APCR Rounds for details. Spotting Scope [Expand][Hide] Cost 25 Time 25 Hotkey S Effects The Spotting Scope increases sight and gun range of the Hetzer Tank Destroyer. ESee Upgrade Spotting Scope for details. Vehicle Weapons 75mm Hetzer [Expand][Hide] Weapon 75mm Hetzer See Weapon 75mm Hetzer for details. 75mm Hetzer APCR [Expand][Hide] Weapon 75mm Hetzer APCR See Weapon 75mm Hetzer APCR for details. MG42 Hetzer [Expand][Hide] Weapon MG42 Hetzer See Weapon MG42 Hetzer for details. Retrieved from http //coh-stats.com/Vehicle Hetzer
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Love, Loss, And What I Wore Algonquin Books?Ilene Beckerman Mother of the Bride Algonquin Books?Ilene Beckerman My Circle A Friends and Family Organizer Chronicle BooksIlene Beckerman Makeovers At The Beauty Counter Of Happiness Algonquin Books?Ilene Beckerman What We Do for Love Algonquin Books?Ilene Beckerman Mother Of The Bride Algonquin Books?Ilene Beckerman Love, Loss, and What I Wore My Life in Fashion Algonquin Books?Ilene Beckerman What We Do for Love Listen & Live Audio?Ilene Beckerman Wenn Toechter erwachsen werden. Nichts ist einfach. Nicht einmal das Glueck. Goldmann Wilhelm GmbH?Ilene Beckerman Was tut man nicht alles aus Liebe! Sanssouci Verlag?Ilene Beckerman Liebe, Leid und welches Kleid? Sanssouci Verlag?Ilene Beckerman Mother of the Bride The Dream, the Reality, the Search for a Perfect Dress Algonquin Books?Ilene Beckerman
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2031 Building a Space Station 問題 http //acm.pku.edu.cn/JudgeOnline/problem?id=2031 解答方針 最小全域木．今回はKruskalのアルゴリズムで解いた． 解答例 import java.io.*; import java.util.*; import java.text.*; public class Main{ public static void main(String[] args)throws IOException{ Scanner sc = new Scanner(System.in); DecimalFormat formatter = new DecimalFormat("#0.000"); int n = sc.nextInt(); while(n!=0){ double data[][] = new double[n][4]; for(int i=0;i n;i++){ data[i][0] = sc.nextDouble(); data[i][1] = sc.nextDouble(); data[i][2] = sc.nextDouble(); data[i][3] = sc.nextDouble(); } Solver s = new Solver(n,data); double ans = s.solve(); System.out.println(formatter.format(ans)); n = sc.nextInt(); } } } class Solver{ int size; int connected[];//expression of MF-SET PriorityQueue Edge q; Solver(int n,double data[][]){ size = n; connected = new int[size]; q = new PriorityQueue Edge (); for(int i=0;i n;i++){ for(int j=i+1;j n;j++){ double elen = edgeLength(data[i],data[j]); Edge e = new Edge(i,j,elen); q.offer(e); } } for(int i=0;i n;i++) connected[i] = i; } public double solve(){ double corlen; corlen = 0; //Kruskal s algorithm while(!q.isEmpty()){ Edge e = q.poll(); int i = e.p1; int j = e.p2; if(connected[i]!=connected[j]){ merge(i,j); corlen += e.length; } } return corlen; } //MERGE OF MF-SET private void merge(int i,int j){ int m1 = connected[i]; int m2 = connected[j]; for(int k=0;k size;k++){ if(connected[k]==m2) connected[k] = m1; } } private double edgeLength(double d1[],double d2[]){ double dx = d1[0]-d2[0]; double dy = d1[1]-d2[1]; double dz = d1[2]-d2[2]; double dist = Math.sqrt(dx*dx+dy*dy+dz*dz); double prelen = dist - (d1[3]+d2[3]); if(prelen =0.0) return prelen; else return 0.0; } } class Edge implements Comparable Edge { int p1; int p2; double length; Edge(int i,int j,double l){ p1 = i; p2 = j; length = l; } public int compareTo(Edge e){ if(length e.length) return 1; else if(length==e.length) return 0; else return -1; } }
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PATH.ライトサイド ＴＯＰ ＧＥＮＥＲＡＬ icon Expertise 説明 効果 Cautious Nature スタンス使用時にグランシングブローボーナス(4P)コンスト増加 (1/2/3/4)%(10/20/30/40) Saber Shackle セーバースロー使用時、スネア＆ルート発生＆確率？(4P) (2/4/6/8)% Reactive Response ダメージを受けた際にアクション回復(2P) (2/4)% Force Clarity ライトセイバーでのセカンドアタックの確立(２回攻撃かな)(1P) (5)% Anticipate Aggression すべてのダメージを軽減(2P) (4/8)% Riposte ５M以内のカウンターアタックの失敗確立なのかな？(2P) (25/50)% Saber Reflect 一定の割合のダメを相手に跳ね返す？(1P) Saber Reflectパリー１０％UP？ Improved Saber Reflect 跳ね返すダメージの割合をUP(3P)跳ね返した時にForce Alacrity発生率UP？ (33/66/100)%(16/32/50)% Perceptive Sentinel PvP時にクリティカルチャンスディフェンス(4P) (1/2/3/4)% Forsake Fear JEDIとそのGPメンバーのアクション回復(1P) 1秒に6%(最大10秒)Forsake Fear Hermetic Touch DOT治療かな？使った後一定時間免疫あり(1P)スタックを取り除けるのかな？ Hermetic Touch Soothing Aura ヒール回復量のUP(4P) (25/50/75/100)% PATH.ダークサイド ＴＯＰ ＧＥＮＥＲＡＬ icon Expertise 説明 効果 Remorseless Nature フォーカス使用時にスタミナ増加(4P) (10/20/30/40) Brutality クリティカルUP？(4P) (1/2/3/4)% Dark Lightning フォースライトニング上位アビ取得？(1)微量のDOTダメージありなの？ Force Lightning Maelstrom 範囲攻撃アビリティー(1P) Maelstrom Wracking Energy スネア時間増加フォースライトニング＆ドレイン＆Maelstromのアーマー貫通？(4P) 修正して Tempt Hatred 与えたダメージでアクション回復(2P) (2/4)% Force Drain ダメージを与えヘルスを吸収(1P) Force Drain Improved Force Drain ドレインの成功率？吸収力？謎(3P) (33/66/100)% Ruthless Precision 攻撃ダメージUP(4P) (2/4/6/8)% Force Choke あなたの相手を破損する軽い窒息攻撃(1P)DOTあり？ Force Choke Improved Force Choke チョークダメージUP(2P)DOTダメージUP？ (10/20)%(10/20)% Strangulation わかりません(2P) 修正して PATH ＴＯＰ ＧＥＮＥＲＡＬ icon Expertise 説明 効果 Cloud Minds マインドトリック上位アビリティー(1P)範囲マインドトリック？ Cloud Minds Expansive Trickery Cloud Mindsの範囲UP(3P) (2/4/6)% Lethargy Cloud Mindsの効果時間増加(4P) (2/4/6/8)秒
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ALL-PERMUTATIONS Arguments list [generic-function] returns a series containing all permutations of list
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SOS1-/- mice impair Cdc42 activation in PC12 cells. SOS-/- fly impair the development of eyes. Overexpression of GAB1 exhibit neurite outgrowth, DNA synthesis and survival in PC12 cells. SHCB-deficient animals exhibit a loss of peptidergic and nonpeptidergic nociceptive sensory neurons, Hippocampal long-term potentiation in ShcC mutant mice is significantly enhanced. SHCA controls the size of brain. Cbl-b null mice exhibit the enhancement of long-term memory. SOS1-/- mice impair Cdc42 activation in PC12 cells. 1 Mol Biol Cell. 2005 May;16(5) 2207-17. Epub 2005 Feb 23. Local phosphatidylinositol 3,4,5-trisphosphate accumulation recruits Vav2 and Vav3 to activate Rac1/Cdc42 and initiate neurite outgrowth in nerve growth factor-stimulated PC12 cells. Aoki K, Nakamura T, Fujikawa K, Matsuda M. Department of Tumor Virology, Research Institute for Microbial Diseases, Osaka University, Osaka 565-0871, Japan. Neurite outgrowth is an important process in the formation of neuronal networks. Rac1 and Cdc42, members of the Rho-family GTPases, positively regulate neurite extension through reorganization of the actin cytoskeleton. Here, we examine the dynamic linkage between Rac1/Cdc42 and phosphatidylinositol 3-kinase (PI3-kinase) during nerve growth factor (NGF)-induced neurite outgrowth in PC12 cells. Activity imaging using fluorescence resonance energy transfer probes showed that PI3-kinase as well as Rac1/Cdc42 was transiently activated in broad areas of the cell periphery immediately after NGF addition. Subsequently, local and repetitive activation of PI3-kinase and Rac1/Cdc42 was observed at the protruding sites. Depletion of Vav2 and Vav3 by RNA interference significantly inhibited both Rac1/Cdc42 activation and the formation of short processes leading to neurite outgrowth. At the NGF-induced protrusions, local phosphatidylinositol 3,4,5-trisphosphate accumulation recruited Vav2 and Vav3 to activate Rac1 and Cdc42, and conversely, Vav2 and Vav3 were required for the local activation of PI3-kinase. These observations demonstrated for the first time that Vav2 and Vav3 are essential constituents of the positive feedback loop that is comprised of PI3-kinase and Rac1/Cdc42 and cycles locally with morphological changes. Publication Types Research Support, Non-U.S. Gov t PMID 15728722 [PubMed - indexed for MEDLINE] SOS-/- fly impair the development of eyes. 1 Cell. 1991 Jan 11;64(1) 39-48. Genetic dissection of a neurodevelopmental pathway Son of sevenless functions downstream of the sevenless and EGF receptor tyrosine kinases. Rogge RD, Karlovich CA, Banerjee U. Department of Biology, University of California, Los Angeles 90024. We have isolated a dominant mutation in a gene called Son of sevenless (Sos) that is an allele-specific suppressor of the sevenless phenotype. This suppressor function is autonomously required in R7 and is sensitive to the dosage of the Sos and bride of sevenless genes. Loss-of-function alleles of Sos are recessive lethals, but in the eye Sos has a role in R cell development. Mutations in Sos also interact with the Ellipse allele of the Drosophila EGF receptor. We propose a model suggesting that the Sos product is downstream of sevenless and the EGF receptor, and that the dominant suppression results from the overexpression or increased activity of the gene product. Publication Types Research Support, Non-U.S. Gov t Research Support, U.S. Gov t, P.H.S. PMID 1846090 [PubMed - indexed for MEDLINE] Overexpression of GAB1 exhibit neurite outgrowth, DNA synthesis and survival in PC12 cells. 1 J Biol Chem. 1999 Dec 24;274(52) 37307-14. Gab1 mediates neurite outgrowth, DNA synthesis, and survival in PC12 cells. Korhonen JM, Said FA, Wong AJ, Kaplan DR. Montreal Neurological Institute, Brain Tumor Research Centre, Montreal, Quebec H3A 2B4, Canada. The Gab1-docking protein has been shown to regulate phosphatidylinositol 3-kinase PI3K activity and potentiate nerve growth factor (NGF)-induced survival in PC12 cells. Here, we investigated the potential of Gab1 to induce neurite outgrowth and DNA synthesis, two other important aspects of NGF-induced neuronal differentiation of PC12 cells and NGF-independent survival. We generated a recombinant adenovirus encoding hemagglutinin (HA)-epitope-tagged Gab1 and expressed this protein in PC12 cells. HA-Gab1 was constitutively tyrosine-phosphorylated in PC12 cells and induced the phosphorylation of Akt/protein kinase B and p44/42 mitogen-activated protein kinase. HA-Gab1-stimulated a 10-fold increase in neurite outgrowth in the absence of NGF and a 5-fold increase in NGF-induced neurite outgrowth. HA-Gab1 also stimulated DNA synthesis and caused NGF-independent survival in PC12 cells. Finally, we found that HA-Gab1-induced neuritogenesis was completely suppressed by pharmacological inhibition of mitogen-activated protein kinase kinase (MEK) activity and 50% suppressed by inhibition of PI3K activity. In contrast, HA-Gab1-stimulated cell survival was efficiently suppressed only by inhibition of both PI3K and MEK activities. These results indicate that Gab1 is capable of mediating differentiation, DNA synthesis, and cell survival and uses both PI3K and MEK signaling pathways to achieve its effects. Publication Types Research Support, Non-U.S. Gov t PMID 10601297 [PubMed - indexed for MEDLINE] SHCB-deficient animals exhibit a loss of peptidergic and nonpeptidergic nociceptive sensory neurons, 1 Neuron. 2000 Dec;28(3) 819-33. The mammalian ShcB and ShcC phosphotyrosine docking proteins function in the maturation of sensory and sympathetic neurons. Sakai R, Henderson JT, O Bryan JP, Elia AJ, Saxton TM, Pawson T. Program in Molecular Biology and Cancer, Samuel Lunenfeld Research Institute, Mount Sinai Hospital, 600 University Avenue, M5G 1X5, Toronto, Ontario, Canada. Shc proteins possess SH2 and PTB domains and serve a scaffolding function in signaling by a variety of receptor tyrosine kinases. There are three known mammalian Shc genes, of which ShcB and ShcC are primarily expressed in the nervous system. We have generated null mutations in ShcB and ShcC and have obtained mice lacking either ShcB or ShcC or both gene products. ShcB-deficient animals exhibit a loss of peptidergic and nonpeptidergic nociceptive sensory neurons, which is not enhanced by additional loss of ShcC. Mice lacking both ShcB and ShcC exhibit a significant loss of neurons within the superior cervical ganglia, which is not observed in either mutant alone. The results indicate that these Shc family members possess both unique and overlapping functions in regulating neural development and suggest physiological roles for ShcB/ShcC in TrkA signaling. PMID 11163269 [PubMed - indexed for MEDLINE] Hippocampal long-term potentiation in ShcC mutant mice is significantly enhanced. 1 J Neurosci. 2005 Feb 16;25(7) 1826-35. Hippocampal synaptic modulation by the phosphotyrosine adapter protein ShcC/N-Shc via interaction with the NMDA receptor. Miyamoto Y, Chen L, Sato M, Sokabe M, Nabeshima T, Pawson T, Sakai R, Mori N. Department of Molecular Genetics, National Institute for Longevity Sciences, Oobu 474-8522, Japan. N-Shc (neural Shc) (also ShcC), an adapter protein possessing two phosphotyrosine binding motifs [PTB (phosphotyrosine binding) and SH2 (Src homology 2) domains], is predominantly expressed in mature neurons of the CNS and transmits neurotrophin signals from the TrkB receptor to the Ras/mitogen-activated protein kinase (MAPK) pathway, leading to cellular growth, differentiation, or survival. Here, we demonstrate a novel role of ShcC, the modulation of NMDA receptor function in the hippocampus, using ShcC gene-deficient mice. In behavioral analyses such as the Morris water maze, contextual fear conditioning, and novel object recognition tasks, ShcC mutant mice exhibited superior ability in hippocampus-dependent spatial and nonspatial learning and memory. Consistent with this finding, electrophysiological analyses revealed that hippocampal long-term potentiation in ShcC mutant mice was significantly enhanced, with no alteration of presynaptic function, and the effect of an NMDA receptor antagonist on its expression in the mutant mice was notably attenuated. The tyrosine phosphorylation of NMDA receptor subunits NR2A and NR2B was also increased, suggesting that ShcC mutant mice have enhanced NMDA receptor function in the hippocampus. These results indicate that ShcC not only mediates TrkB-Ras/MAPK signaling but also is involved in the regulation of NMDA receptor function in the hippocampus via interaction with phosphotyrosine residues on the receptor subunits and serves as a modulator of hippocampal synaptic plasticity underlying learning and memory. PMID 15716419 [PubMed - indexed for MEDLINE] SHCA controls the size of brain. 1 J Neurosci. 2006 Jul 26;26(30) 7885-97. Neural-specific inactivation of ShcA results in increased embryonic neural progenitor apoptosis and microencephaly. McFarland KN, Wilkes SR, Koss SE, Ravichandran KS, Mandell JW. Department of Pathology (Neuropathology), Beirne B. Carter Center for Immunology Research, University of Virginia, Charlottesville, Virginia 22908, USA. Brain size is precisely regulated during development and involves coordination of neural progenitor cell proliferation, differentiation, and survival. The adapter protein ShcA transmits signals from receptor tyrosine kinases via MAPK (mitogen-activated protein kinase)/ERK (extracellular signal-regulated kinase) and PI3K (phosphatidylinositol 3-kinase)/Akt signaling pathways. In the CNS, ShcA expression is high during embryonic development but diminishes as cells differentiate and switches to ShcB/Sck/Sli and ShcC/N-Shc/Rai. To directly test ShcA function in brain development, we used Cre/lox technology to express a dominant-negative form of ShcA (ShcFFF) in nestin-expressing neural progenitors. ShcFFF-expressing mice display microencephaly with brain weights reduced to 50% of littermate controls throughout postnatal and adult life. The cerebrum appeared most severely affected, but the gross architecture of the brain is normal. Body weight was mildly affected with a delay in reaching mature weight. At a mechanistic level, the ShcFFF microencephaly phenotype appears to be primarily attributable to elevated apoptosis levels throughout the brain from embryonic day 10.5 (E10.5) to E12, which declined by E14.5. Apoptosis remained at normal basal levels throughout postnatal development. Proliferation indices were not significantly altered in the embryonic neuroepithelium or within the postnatal subventricular zone. In another approach with the same nestin-Cre transgene, conditional deletion of ShcA in mice with a homozygous floxed shc1 locus also showed a similar microencephaly phenotype. Together, these data suggest a critical role for ShcA in neural progenitor survival signaling and in regulating brain size. PMID 16870734 [PubMed - indexed for MEDLINE] Cbl-b null mice exhibit the enhancement of long-term memory. 1 Proc Natl Acad Sci U S A. 2006 Mar 28;103(13) 5125-30. Epub 2006 Mar 20. Enhancement of long-term memory retention and short-term synaptic plasticity in cbl-b null mice. Tan DP, Liu QY, Koshiya N, Gu H, Alkon D. Blanchette Rockefeller Neurosciences Institute, Rockville, MD 20850, USA. dptan@brni-jhu.org The cbl-b gene is a member of the cbl protooncogene family. It encodes a protein with multiple domains, which can interact with other proteins in a variety of signaling pathways. The functions of cbl family genes in the brain are unknown. In this report, we used genetic, immunohistochemical, behavioral, and electrophysiological approaches to study the role of cbl-b in learning and memory. Cbl-b null mice developed normally and had no abnormalities in their locomotor performance. In spatial learning and memory studies, cbl-b null and WT mice performed similarly during training. To test memory retention, two probe trials were used. cbl-b null mice performed slightly better 1 day after training. However, in the probe trial 45 days after training, the cbl-b null group showed significantly higher memory retention than WT mice, suggesting an enhancement of long-term memory. Using electrophysiological approaches, we found there was enhanced paired-pulse facilitation in the Schaffer Collateral-CA1 glutamatergic synapses of the cbl-b null mice. On the other hand, there was no difference in long-term potentiation between the two groups of mice. In summary, we provide evidence that (i) cbl-b protein is concentrated in the synaptic regions of CA1, CA3, and the dentate gyrus of the hippocampus; (ii) cbl-b null mice have enhanced long-term memory; and (iii) cbl-b null mice show an enhancement in short-term plasticity. These results indicate that cbl-b is a negative regulator of long-term memory, and its neuronal mechanism regulates synaptic transmission in the hippocampus. PMID 16549761 [PubMed - indexed for MEDLINE]
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Nanhai county is the most Sexy Lingeri southern mist state, a piece of land of confusion. Huang Siqin is not the eagle wolf guard dedicated White Eagle head, said he to fix the collusion, no investigation, this is a well known secret in South xinjiang. This place in Qingzhou is a great sin, but in a fog state what is not. "Agreed to his request!"... As a result, Li Qingshan has incredible speed to get promoted, becoming the county in the South China Sea a new term of the White Eagle command. Sexy Lingeri "Hey, officials, murder and burn by amnesty!" Li Qingshan put on a white eagle leading fashion, chic straight, waist also hung a small Silver Eagle, smiled to Ann said. And then waiting for various people to kill, ready to enjoy the sea native enthusiasm. But he left wait right etc, but even the enemy s shadow not wait, even that he was killed by white Langwei master, did not come for the disciples pay off old scores, days before it than in Qingzhou also peace. Li Qingshan was hard to understand, Sexy Lingeri it is dangerous in South? Feel like what is wrong, and to Chenghai Hou house. "Congratulations to guide, become white eagle command. I do not know this day come, what can I do for you?" Chenghai climate with three points carefully asked. "Mayor, you say, why did no one come to kill me?" Li Qingshan not polite to sit down, frowning asked. Chenghai Hou was stunned, what is the problem? Also a very distressed look, with great care and asked "why people want to kill you?" "You see, I was born the day door upright, also said to mop up the devils, in addition to the traditional moral principles, Chang Shun me, against me dead, these words should Sexy Lingeri be passed out! Those magic fix us not to kill me, what are you waiting for?" "Do you want them to kill you?" Chenghai Hou fantastic way. "Of course, or I said what do those words? Oh, that make you feel shy!" "This is to what?" Chenghai waiting to see Li Qingshan s eyes, like a monster, originally he deliberately said the words, just to taunt South County fix, attract hatred to kill him. This is what? "I want to kill! Nor is man, what you want." Li Qingshan frankly, now, he doesn t want to conceal their Sexy Lingeri true purpose, besides what justice crap. He came to a major purpose of Nanhai county is a good war! Let the tiger demon killing get the most satisfied... "Isn t it the first precept of Buddhism killing?" Chenghai Hou Leng for a while. Li Qingshan hey hey a smile "in addition to the traditional moral principles, in addition to the traditional moral principles!" Chenghai Hou wanted to say, you don t need to explain, you are in Denon monastery to suppress bad! Others considered dangerous Jedi Sexy Lingeri in southern Xinjiang, is he to find the place. He gave a long sigh "I m afraid temporarily not to kill you!" Why? I heard very much Huang Siqin not friends? Isn t there a handout spirit?" Chenghai Hou cough twice, he is also one of Huang Siqin s "friends". After an attack by innuendo, Li Qingshan also finally understand, why people don t come looking for his trouble. The first is that he killed Huang Siqin to show when the powerful actual strength, have been enough to deter most fix. Huang Siqin have a network of relationships, but died as.Side of Zhu Lian, also feel happy for him. "My father, that captured the Bauhinia Ling Kou, but my dear in the prince." Zhao Chenxiao happily to the credit for Zhao Huan, let Zhao Huan is feeling happy. Sexy Lingeri Yeah, this one battle, dear army led by Prince when power! The prince made dear in the military exploits, the prince is Prince exploits; the military exploits, also it is to his credit. "Never think, this jade butcher is really a brave warrior. A snowstorm to surprise the Bauhinia Ling Kou, beheaded nearly 1000, was the first work piece. Good, good..." Zhao Huan wanted to think, sink a track "since it is the first work piece, can t maltreat him. So, he ordered him to be a tan state so that the militia, Sexy Lingeri - https //www.amoretu.com/sexy-lingerie sage think that how?" Zhu Lian Yi Zheng, a frown frown. "Tanzhou tuanlian make, some of them." So that the militia is a virtual level, not the state troops and horses is a palm, from the military attache two products. Yu Yin now, but is a goods from the six, one to tuanlian make, it is since Song Dynasty, there has never been a thing. Zhao Huan this just responds to come over, the actual office Yu yin. Fortunately, this is at home in front of the person lost face, if in the hall say such words, less by courtiers against. The present awkward smile, Zhao Huan hesitated, light tone way "the sages thought, when in what position?" Zhu Lian smiled and said "within the palace without intervention, the minister Qie also just freely so that." "The sage is casual, not to be taken seriously." The side of Zhao Chen s vision, shows some hope, let Zhu Lian also some difficult. She certainly knows, Zhao Chen is what the mind. Yu Yin is Zhao Chen, natural hope Yu Yin can benefit. The problem is, Yu AMORETU Yin Prince dear in the control, this does not enter the grade, his real office, or Hyobu doctor, is not what the magnifico. At the outset by Huang Chang, Yu Yin got a hereditary. But in the end, he did not fame, even made illustrious military exploits, but also may not be Keju origin... Hesitated for a long time, Zhu Lian whispered "the minister Qie remembers, Jizhou there situation is very confusing, why not follow the example of other places, set up patrol division? For example, Jizhou Xunjiansi, let Yu Xiaoyi do a patrol, also is the reward of his service.
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YY-CHR BB English Translation Translation of the BB thread dedicated to user requests and bug reports. Between 2009 and 2022, close to 200 posts were made. waiwai ：2009/01/05(月) 22 32 54 This is the thread for reporting bugs and posting requests for YY-CHR Graphics Editor For Use With Retro Game Consoles. You can getthe latests version from here ttp //www.geocities.jp/yy_6502/0yychr.html waiwai ：2009/01/05(月) 23 02 22 What i want to deal with in the near future Support for the 4BPP SMS/GG/WSC image format Support for .col palette set files Image format plugin conversion
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Radiation 概要 Considered by many to be unethical, the radiation turret isn t favored by many of the executive officers of the fleet. A shielded dome opens up to reveal the core of an overloading nuclear reactor, with the radiation being blown out onto the enemy. The radiation causes the enemy to be weakened, making all other turrets more effective when firing at these irradiated enemies. Radiation dosing can be increased bu higher density coverage, making tight clusters of radiation turrets effective for a choke point. タレットユニーク効果 近くの的に照射し、他のダメージに対して弱くさせる。 ダメージタイプ Heat 熱ダメージ 使用オフィサー Null レベルアップによる威力とコスト レベル 射程距離 威力 資源コスト 1 30 86 20 2 35 343 83 3 40 1088 263